Viral binding-induced signaling drives a unique and extended intracellular trafficking pattern during infection of primary monocytes.
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LPS promotes a monocyte phenotype permissive for human cytomegalovirus immediate-early gene expression upon infection but not reactivation from latencyHuman Cytomegalovirus Requires Epidermal Growth Factor Receptor Signaling To Enter and Initiate the Early Steps in the Establishment of Latency in CD34+ Human Progenitor Cells.HCMV utilizes a non-traditional STAT1 activation cascade via signaling through EGFR and integrins to efficiently promote the motility, differentiation, and polarization of infected monocytes.Navigating the Host Cell Response during Entry into Sites of Latent Cytomegalovirus Infection.HCMV Infection and Apoptosis: How Do Monocytes Survive HCMV Infection?
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Viral binding-induced signaling drives a unique and extended intracellular trafficking pattern during infection of primary monocytes.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 July 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Viral binding-induced signalin ...... nfection of primary monocytes.
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Viral binding-induced signalin ...... nfection of primary monocytes.
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type
label
Viral binding-induced signalin ...... nfection of primary monocytes.
@en
Viral binding-induced signalin ...... nfection of primary monocytes.
@nl
prefLabel
Viral binding-induced signalin ...... nfection of primary monocytes.
@en
Viral binding-induced signalin ...... nfection of primary monocytes.
@nl
P2093
P2860
P356
P1476
Viral binding-induced signalin ...... nfection of primary monocytes.
@en
P2093
Donna Collins-McMillen
Jung Heon Kim
Patrizia Caposio
P2860
P304
P356
10.1073/PNAS.1604317113
P407
P577
2016-07-18T00:00:00Z