Positive role of CCAAT/enhancer-binding protein homologous protein, a transcription factor involved in the endoplasmic reticulum stress response in the development of colitis - sprookjes - yvandenbroek - TriplyDB

Positive role of CCAAT/enhancer-binding protein homologous protein, a transcription factor involved in the endoplasmic reticulum stress response in the development of colitis

Positive role of CCAAT/enhancer-binding protein homologous protein, a transcription factor involved in the endoplasmic reticulum stress response in the development of colitis

http://www.wikidata.org/entity/Q37164215

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Endoplasmic reticulum stress-activated C/EBP homologous protein enhances nuclear factor-kappaB signals via repression of peroxisome proliferator-activated receptor gamma CHOP potentially co-operates with FOXO3a in neuronal cells to regulate PUMA and BIM expression in response to ER stress Role of endoplasmic reticulum stress and autophagy as interlinking pathways in the pathogenesis of inflammatory bowel disease RNase L controls terminal adipocyte differentiation, lipids storage and insulin sensitivity via CHOP10 mRNA regulation Triglyceride-rich lipoprotein modulates endothelial vascular cell adhesion molecule (VCAM)-1 expression via differential regulation of endoplasmic reticulum stress Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress Prevention of UVB radiation-induced epidermal damage by expression of heat shock protein 70.Expression and function of HSP110 family in mouse testis after vasectomy Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease.Melatonin Modulates Neuronal Cell Death Induced by Endoplasmic Reticulum Stress under Insulin Resistance Condition.Regulation of basal cellular physiology by the homeostatic unfolded protein response Toll-like receptors in inflammatory bowel diseases: a decade later.The stress-response sensor chop regulates the function and accumulation of myeloid-derived suppressor cells in tumors Suppression of expression of endoplasmic reticulum chaperones by Helicobacter pylori and its role in exacerbation of non-steroidal anti-inflammatory drug-induced gastric lesions.Glutamine treatment attenuates endoplasmic reticulum stress and apoptosis in TNBS-induced colitis PKR-dependent CHOP induction limits hyperoxia-induced lung injury.Opposing effects of monomeric and pentameric C-reactive protein on endothelial progenitor cells.Epithelial Sel1L is required for the maintenance of intestinal homeostasis Endoplasmic reticulum stress and inflammation: mechanisms and implications in diabetic retinopathy.Two in-and-out modulation strategies for endoplasmic reticulum stress-linked gene expression of pro-apoptotic macrophage-inhibitory cytokine 1.Endoplasmic Reticulum Stress and Unfolded Protein Response Pathways: Potential for Treating Age-related Retinal Degeneration cEBP Homologous Protein Expression in Macrophages Regulates the Magnitude and Duration of IL-6 Expression and Dextran Sodium Sulfate Colitis Bifidobacteria Prevent Tunicamycin-Induced Endoplasmic Reticulum Stress and Subsequent Barrier Disruption in Human Intestinal Epithelial Caco-2 Monolayers The unfolded protein response and gastrointestinal disease Chop Deficiency Protects Mice Against Bleomycin-induced Pulmonary Fibrosis by Attenuating M2 Macrophage Production.The unfolded protein response and chemical chaperones reduce protein misfolding and colitis in mice.C/EBP homologous protein (CHOP) deficiency ameliorates renal fibrosis in unilateral ureteral obstructive kidney disease.Ribosomal alteration-derived signals for cytokine induction in mucosal and systemic inflammation: noncanonical pathways by ribosomal inactivation.Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance.ER stress and the unfolded protein response in intestinal inflammation.Endoplasmic Reticulum Stress PERK-ATF4-CHOP Pathway Is Associated with Hypothalamic Neuronal Injury in Different Durations of Stress in Rats.Signalling danger: endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation.Integrated stress response-altered pro-inflammatory signals in mucosal immune-related cells.Endoplasmic reticulum stress in intestinal epithelial cell function and inflammatory bowel disease Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.Endoplasmic Reticulum Stress and Oxidative Stress: A Vicious Nexus Implicated in Bowel Disease Pathophysiology Crosstalk between endoplasmic reticulum stress and oxidative stress in apoptosis induced by α-tocopheryl succinate in human gastric carcinoma cells.The involvement of endoplasmic reticulum stress in flavonoid-induced protection on cardiac cell death caused by ischaemia/reperfusion.C/EBP homologous protein modulates liraglutide-mediated attenuation of non-alcoholic steatohepatitis.C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation.

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Positive role of CCAAT/enhancer-binding protein homologous protein, a transcription factor involved in the endoplasmic reticulum stress response in the development of colitis

http://www.wikidata.org/entity/Q37164215

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 April 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Positive role of CCAAT/enhance ...... in the development of colitis

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Positive role of CCAAT/enhance ...... in the development of colitis.

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Positive role of CCAAT/enhance ...... in the development of colitis

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Positive role of CCAAT/enhance ...... in the development of colitis.

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prefLabel

Positive role of CCAAT/enhance ...... in the development of colitis

@en

Positive role of CCAAT/enhance ...... in the development of colitis.

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AJPATH.2009.080864

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The American Journal of Pathology

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Positive role of CCAAT/enhance ...... in the development of colitis

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Keizo Sato

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Ken-Ichiro Tanaka

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Motoyoshi Endo

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Takushi Namba

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Tatsuya Hoshino

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Tohru Mizushima

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Tomoaki Ishihara

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Tomomi Gotoh

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Yosuke Ito

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P2860

Inflammatory Bowel Disease

TRB3, a novel ER stress-inducible gene, is induced via ATF4-CHOP pathway and is involved in cell death

Roles of CHOP/GADD153 in endoplasmic reticulum stress

Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

hsp70-DnaJ chaperone pair prevents nitric oxide- and CHOP-induced apoptosis by inhibiting translocation of Bax to mitochondria

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Tuberculosis associated with infliximab, a tumor necrosis factor alpha-neutralizing agent

Nitric oxide-induced apoptosis in RAW 264.7 macrophages is mediated by endoplasmic reticulum stress pathway involving ATF6 and CHOP

Increased sensitivity to dextran sodium sulfate colitis in IRE1beta-deficient mice

CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

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1786-1798

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scholarly article

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10.2353/AJPATH.2009.080864

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5

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174

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2009-04-09T00:00:00Z

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24271103

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19359519

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endoplasmic reticulum

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2671267

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