Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung. - sprookjes - yvandenbroek - TriplyDB

Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung.

Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung.

http://www.wikidata.org/entity/Q37278959

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Low-dose arsenic compromises the immune response to influenza A infection in vivo Lung tumors in mice induced by "whole-life" inorganic arsenic exposure at human-relevant doses Effects of low-dose drinking water arsenic on mouse fetal and postnatal growth and development Permeation of roxarsone and its metabolites increases caco-2 cell proliferation In utero arsenic exposure and fetal immune repertoire in a US pregnancy cohort Arsenic in eggs and excreta of laying hens in Bangladesh: a preliminary study In utero arsenic exposure and infant infection in a United States cohort: a prospective study Chronic arsenic exposure in nanomolar concentrations compromises wound response and intercellular signaling in airway epithelial cells A novel approach to analyze gene expression data demonstrates that the DeltaF508 mutation in CFTR downregulates the antigen presentation pathway.Prenatal arsenic exposure and the epigenome: altered microRNAs associated with innate and adaptive immune signaling in newborn cord blood.Exposure to multiple metals from groundwater-a global crisis: geology, climate change, health effects, testing, and mitigation.Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?Evidence from Chile that arsenic in drinking water may increase mortality from pulmonary tuberculosis Arsenic immunotoxicity: a review.Arsenic alters ATP-dependent Ca²+ signaling in human airway epithelial cell wound response.Genetic susceptible locus in NOTCH2 interacts with arsenic in drinking water on risk of type 2 diabetes.Arsenic compromises conducting airway epithelial barrier properties in primary mouse and immortalized human cell cultures.Environmental arsenic exposure and microbiota in induced sputum.Arsenic exposure and prevalence of the varicella zoster virus in the United States: NHANES (2003-2004 and 2009-2010)Expression of arsenic resistance genes in the obligate anaerobe Bacteroides vulgatus ATCC 8482, a gut microbiome bacterium.Arsenic promotes ubiquitinylation and lysosomal degradation of cystic fibrosis transmembrane conductance regulator (CFTR) chloride channels in human airway epithelial cells Arsenic exposure is associated with pediatric pneumonia in rural Bangladesh: a case control study.Differential DNA methylation in umbilical cord blood of infants exposed to low levels of arsenic in utero.A Systems Toxicology-based Approach Reveals Biological Pathways Dysregulated by Prenatal Arsenic Exposure.Effects of health intervention programs and arsenic exposure on child mortality from acute lower respiratory infections in rural Bangladesh.Arsenic Exposure and Immunotoxicity: a Review Including the Possible Influence of Age and Sex.Arsenic-contaminated freshwater: assessing arsenate and arsenite toxicity and low-dose genotoxicity in Gammarus elvirae (Crustacea; Amphipoda).Activating transcription factor 4 underlies the pathogenesis of arsenic trioxide-mediated impairment of macrophage innate immune functions.Unfolded protein response (UPR) signaling regulates arsenic trioxide-mediated macrophage innate immune function disruption.Sodium arsenite represses the expression of myogenin in C2C12 mouse myoblast cells through histone modifications and altered expression of Ezh2, Glp, and Igf-1.AIM2 inflammasome mediates Arsenic-induced secretion of IL-1 β and IL-18.Arsenic exposure impels CD4 commitment in thymus and suppress T cell cytokine secretion by increasing regulatory T cells.Arsenic alters transcriptional responses to Pseudomonas aeruginosa infection and decreases antimicrobial defense of human airway epithelial cells.Effects of arsenic on porcine dendritic cells in vitro.Inorganic arsenic can be potent granulotoxin in mammalian neutrophils in vitro.STAT3 in arsenic lung carcinogenicity.Health effects following subacute exposure to geogenic dust collected from active drainage surfaces (Nellis Dunes Recreation Area, Las Vegas, NV).Arsenic handicap? Prenatal exposure worsens influenza infections in young mice.Pathological, immunological and biochemical markers of subchronic arsenic toxicity in rats.Chronic arsenic intoxication diagnostic score (CAsIDS).

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Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung.

http://www.wikidata.org/entity/Q37278959

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scientific article published on 04 March 2009

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Chronic exposure to arsenic in ...... response genes in mouse lung.

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Chronic exposure to arsenic in ...... response genes in mouse lung.

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Environmental Health Perspectives

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Chronic exposure to arsenic in ...... e response genes in mouse lung

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Athena P Nomikos

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Courtney D Kozul

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Daniel J Weiss

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Jennifer C Davey

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Jessica E Thorpe

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Joshua W Hamilton

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Julie A Gosse

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Michael A Ihnat

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Phillip L Eisenhauer

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P2860

Cancer risks from arsenic in drinking water

Toll-like receptor signalling

Genomic and proteomic profiling of responses to toxic metals in human lung cells.

Gene expression of inflammatory molecules in circulating lymphocytes from arsenic-exposed human subjects.

ADAMs: key components in EGFR signalling and development

Sodium arsenate induces overproduction of interleukin-1alpha in murine keratinocytes: role of mitochondria

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis

Increased mortality from lung cancer and bronchiectasis in young adults after exposure to arsenic in utero and in early childhood

Sodium arsenite exposure alters cell migration, focal adhesion localization and decreases tyrosine phosphorylation of focal adhesion kinase in H9C2 myoblasts

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1108-1115

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scholarly article

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10.1289/EHP.0800199

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7

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117

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Thomas H Hampton

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2009-03-04T00:00:00Z

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26717125

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19654921

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2717138

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