Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors. - sprookjes - yvandenbroek - TriplyDB

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

http://www.wikidata.org/entity/Q37368169

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MicroRNA profiling of ovarian granulosa cell tumours reveals novel diagnostic and prognostic markers.SMAD3 Activation: A Converging Point of Dysregulated TGF-Beta Superfamily Signaling and Genetic Aberrations in Granulosa Cell Tumor Development?Improving oocyte quality by transfer of autologous mitochondria from fully grown oocytes.Disruption of postnatal folliculogenesis and development of ovarian tumor in a mouse model with aberrant transforming growth factor beta signaling.Ovarian Cancers: Genetic Abnormalities, Tumor Heterogeneity and Progression, Clonal Evolution and Cancer Stem Cells.The molecular mechanism of ovarian granulosa cell tumors.MicroRNA-10a promotes granulosa cells tumor development via PTEN-AKT/Wnt regulatory axis

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Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

http://www.wikidata.org/entity/Q37368169

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article científic

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article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 09 May 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

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Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

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label

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

@en

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

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Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

@en

Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

@nl

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0008-5472.CAN-15-3358

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Cancer Research

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Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.

@en

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Adrian A Suarez

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Katherine Ebbert

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Kelly A Whelan

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Marilia H Cordeiro

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Megan M Romero

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So-Youn Kim

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P2860

The FOXL2 C134W mutation is characteristic of adult granulosa cell tumors of the ovary.

Mechanisms of metabolic dysfunction in cancer-associated cachexia

Smad1-Smad5 ovarian conditional knockout mice develop a disease profile similar to the juvenile form of human granulosa cell tumors.

FOXO1/3 and PTEN Depletion in Granulosa Cells Promotes Ovarian Granulosa Cell Tumor Development.

Cell autonomous phosphoinositide 3-kinase activation in oocytes disrupts normal ovarian function through promoting survival and overgrowth of ovarian follicles.

Granulosa cell tumor of the ovary: 10 years follow-up data of 65 patients.

Activating mutations of the stimulatory g protein in juvenile ovarian granulosa cell tumors: a new prognostic factor?

Gonadotropin-positive pituitary tumors accompanied by ovarian tumors in aging female ERbeta-/- mice.

The emerging mutational landscape of G proteins and G-protein-coupled receptors in cancer.

Oocyte-expressed TGF-beta superfamily members in female fertility.

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3851-3861

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scholarly article

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10.1158/0008-5472.CAN-15-3358

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13

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76

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Teresa K. Woodruff

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2016-05-09T00:00:00Z

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27197196

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5081136

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