Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
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Indomethacin-induced apoptosis in esophageal adenocarcinoma cells involves upregulation of Bax and translocation of mitochondrial cytochrome C independent of COX-2 expressionFAS/FASL expression profile as a prognostic marker in squamous cell carcinoma of the oral cavity.Cytoplasmic overexpression of CD95L in esophageal adenocarcinoma cells overcomes resistance to CD95-mediated apoptosis.Pigment epithelial-derived factor (PEDF)-triggered lung cancer cell apoptosis relies on p53 protein-driven Fas ligand (Fas-L) up-regulation and Fas protein cell surface translocation.Fas receptor (CD95) & Fas ligand (CD178) expression in patients with tobacco-related intraoral squamous cell carcinoma.Alterations of Fas (Apo-1/CD95) gene in cutaneous malignant melanoma.Relationship of Fas, FasL, p53 and bcl-2 expression in human non-small cell lung carcinomas.Streptococcal pyrogenic exotoxin B-induced apoptosis in A549 cells is mediated through alpha(v)beta(3) integrin and Fas.Conformation and free energy analyses of the complex of calcium-bound calmodulin and the Fas death domainTNF-alpha sensitizes normal and fibrotic human lung fibroblasts to Fas-induced apoptosis.Tissue microarray analysis of Fas and FasL expressions in human non-small cell lung carcinomas; with reference to the p53 and bcl-2 overexpressions.Prognostic significance of Fas expression in retinoblastoma.Significance of Fas protein in squamous cell carcinoma of the larynx.Transitional cell carcinoma expresses high levels of Fas ligand in vivo.{2-[(3-Carboxy-1-oxopropyl) amino]-2-deoxy-D-Glucose} suppresses proliferation and induces apoptosis in the human esophageal cancer cell line.Ionizing radiation suppresses FAP-1 mRNA level in A549 cells via p53 activation.Role of Fas and granule exocytosis pathways in tumor-infiltrating T lymphocyte-induced apoptosis of autologous human lung-carcinoma cells.An endoplasmic reticulum-specific stress-activated caspase (caspase-12) is implicated in the apoptosis of A549 epithelial cells by respiratory syncytial virus.Interleukin-1 protects transformed keratinocytes from tumor necrosis factor-related apoptosis-inducing ligand- and CD95-induced apoptosis but not from ultraviolet radiation-induced apoptosis.Trifluoperazine regulation of calmodulin binding to Fas: a computational study.Radiation-induced expression of functional Fas ligand in EBV-positive human nasopharyngeal carcinoma cells.IFN-gammaupregulates apoptosis-related molecules and enhances Fas-mediated apoptosis in human cholangiocarcinoma.Fas (CD95/APO-1) and Fas ligand expression in normal pancreas and pancreatic tumors. Implications for immune privilege and immune escape.Alterations of Fas (Apo-1/CD95) gene in non-small cell lung cancer.Alterations of Fas (APO-1/CD 95) gene and its relationship with p53 in non small cell lung cancer
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P2860
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@en
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@nl
type
label
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@en
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@nl
prefLabel
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@en
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@nl
P2093
P2860
P356
P1476
Lack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.
@en
P2093
P2860
P304
P356
10.1172/JCI1692
P407
P577
1998-03-01T00:00:00Z