Dual FGF-2 and intergrin alpha5beta1 signaling mediate GRAF-induced RhoA inactivation in a model of breast cancer dormancy.
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Dynamic interplay between adhesion surfaces in carcinomas: Cell-cell and cell-matrix crosstalkBreast cancer at bone metastatic sites: recent discoveries and treatment targets.Breast cancer stem cells survive periods of farnesyl-transferase inhibitor-induced dormancy by undergoing autophagy.An In Vitro Dormancy Model of Estrogen-sensitive Breast Cancer in the Bone Marrow: A Tool for Molecular Mechanism Studies and Hypothesis GenerationSMAD signaling and redox imbalance cooperate to induce prostate cancer cell dormancy.Farnesyl transferase inhibitor treatment of breast cancer cells leads to altered RhoA and RhoC GTPase activity and induces a dormant phenotype.A liver microphysiological system of tumor cell dormancy and inflammatory responsiveness is affected by scaffold properties.Reawakening of dormant estrogen-dependent human breast cancer cells by bone marrow stroma secretory senescence
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P2860
Dual FGF-2 and intergrin alpha5beta1 signaling mediate GRAF-induced RhoA inactivation in a model of breast cancer dormancy.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@en
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@nl
type
label
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@en
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@nl
prefLabel
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@en
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@nl
P2860
P1433
P1476
Dual FGF-2 and intergrin alpha ...... del of breast cancer dormancy.
@en
P2093
Judith Barrios
Robert Wieder
P2860
P2888
P356
10.1007/S12307-009-0019-6
P577
2009-03-18T00:00:00Z