MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
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Involvement of AF1q/MLLT11 in the progression of ovarian cancerAF1q Mediates Tumor Progression in Colorectal Cancer by Regulating AKT Signaling.Novel Src/Abl tyrosine kinase inhibitor bosutinib suppresses neuroblastoma growth via inhibiting Src/Abl signaling.Receptor tyrosine kinases (RTKs) in breast cancer: signaling, therapeutic implications and challenges.Aryl Hydrocarbon Receptor Promotes IL-10 Expression in Inflammatory Macrophages Through Src-STAT3 Signaling PathwaySerum cytokine levels in breast cancer patients during neoadjuvant treatment with bevacizumabNovel AF1q/MLLT11 favorably affects imatinib resistance and cell survival in chronic myeloid leukemia
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MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@en
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@nl
type
label
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@en
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@nl
prefLabel
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@en
MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@nl
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MLLT11/AF1q boosts oncogenic STAT3 activity through Src-PDGFR tyrosine kinase signaling.
@en
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Donald M Miller
Joongho Joh
Ju-Hsien Chao
Kevin D Bunting
Maxwell M Krem
Scot C Remick
Shotaro Hagiwara
Soojin Kim
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P304
43960-43973
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10.18632/ONCOTARGET.9759
P407
P577
2016-06-01T00:00:00Z