Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury. - sprookjes - yvandenbroek - TriplyDB

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

http://www.wikidata.org/entity/Q37700810

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.Coagulopathy in the Setting of Mild Traumatic Brain Injury: Truths and Consequences Extracellular vesicles: nature's nanoparticles for improving gene transfer with adeno-associated virus vectors.Omega-3 polyunsaturated fatty acid supplementation attenuates microglial-induced inflammation by inhibiting the HMGB1/TLR4/NF-κB pathway following experimental traumatic brain injury.Acute inhalation of combustion smoke triggers neuroinflammation and persistent anxiety-like behavior in the mouse.Extracellular Vesicles: Packages Sent With Complement.An Inflammation-Centric View of Neurological Disease: Beyond the Neuron.Innate immune responses to trauma.Inhibition of microRNA-155 modulates endotoxin tolerance by upregulating suppressor of cytokine signaling 1 in microglia.Inflammation leads to distinct populations of extracellular vesicles from microglia.Deconvolution of Human Brain Cell Type Transcriptomes Unraveled Microglia-Specific Potential Biomarkers.Omega-3 polyunsaturated fatty acid attenuates the inflammatory response by modulating microglia polarization through SIRT1-mediated deacetylation of the HMGB1/NF-κB pathway following experimental traumatic brain injury.Valproic Acid Attenuates Traumatic Brain Injury-Induced Inflammation in Vivo: Involvement of Autophagy and the Nrf2/ARE Signaling Pathway.Current understanding of neuroinflammation after traumatic brain injury and cell-based therapeutic opportunities.Bidirectional Microglia-Neuron Communication in Health and Disease Extracellular vesicles are increased in the serum of children with autism spectrum disorder, contain mitochondrial DNA, and stimulate human microglia to secrete IL-1β Dibutyryl Cyclic Adenosine Monophosphate Rescues the Neurons From Degeneration in Stab Wound and Excitotoxic Injury Models Multicellular Crosstalk Between Exosomes and the Neurovascular Unit After Cerebral Ischemia. Therapeutic Implications

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Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

http://www.wikidata.org/entity/Q37700810

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 15 March 2017

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@en

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@nl

type

label

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@en

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@nl

prefLabel

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@en

Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.

@nl

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S12974-017-0819-4

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Journal of Neuroinflammation

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Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury

@en

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Alan I Faden

http://www.w3.org/2001/XMLSchema#string

Alok Kumar

http://www.w3.org/2001/XMLSchema#string

Asit Kumar

http://www.w3.org/2001/XMLSchema#string

Bogdan A Stoica

http://www.w3.org/2001/XMLSchema#string

Gelareh Abulwerdi

http://www.w3.org/2001/XMLSchema#string

Ming Yang

http://www.w3.org/2001/XMLSchema#string

Niaz Khan

http://www.w3.org/2001/XMLSchema#string

Stephen R Thom

http://www.w3.org/2001/XMLSchema#string

P2860

P2Y(12) receptors in platelets and other hematopoietic and non-hematopoietic cells

Routes and mechanisms of extracellular vesicle uptake

Depletion of microglia and inhibition of exosome synthesis halt tau propagation.

Targeting miR-155 restores abnormal microglia and attenuates disease in SOD1 mice.

Exosome-mediated transfer of mRNAs and microRNAs is a novel mechanism of genetic exchange between cells

Astrocyte-derived ATP induces vesicle shedding and IL-1 beta release from microglia

In vivo inhibition of miR-155 significantly alters post-stroke inflammatory response

ATP mediates rapid microglial response to local brain injury in vivo

MicroRNAs bind to Toll-like receptors to induce prometastatic inflammatory response

Microparticles generated by decompression stress cause central nervous system injury manifested as neurohypophysial terminal action potential broadening

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s12974-017-0819-4

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47

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scholarly article

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10.1186/S12974-017-0819-4

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1

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2017-03-15T00:00:00Z

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traumatic brain injury

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5351060

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