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A role of melanin-concentrating hormone producing neurons in the central regulation of paradoxical sleepRole of MCH neurons in paradoxical (REM) sleep controlThe supramammillary nucleus and the claustrum activate the cortex during REM sleepLocalization of the brainstem GABAergic neurons controlling paradoxical (REM) sleepA very large number of GABAergic neurons are activated in the tuberal hypothalamus during paradoxical (REM) sleep hypersomniaMajor impairments of glutamatergic transmission and long-term synaptic plasticity in the hippocampus of mice lacking the melanin-concentrating hormone receptor-1Role of the lateral paragigantocellular nucleus in the network of paradoxical (REM) sleep: an electrophysiological and anatomical study in the rat.The endogenous somnogen adenosine excites a subset of sleep-promoting neurons via A2A receptors in the ventrolateral preoptic nucleus.Tuberal hypothalamic neurons secreting the satiety molecule Nesfatin-1 are critically involved in paradoxical (REM) sleep homeostasis.The inhibition of the dorsal paragigantocellular reticular nucleus induces waking and the activation of all adrenergic and noradrenergic neurons: a combined pharmacological and functional neuroanatomical study.Unsupervised online classifier in sleep scoring for sleep deprivation studies.Brainstem structures responsible for paradoxical sleep onset and maintenance.Paradoxical (REM) sleep genesis: the switch from an aminergic-cholinergic to a GABAergic-glutamatergic hypothesis.Levels of Interference in Long and Short-Term Memory Differentially Modulate Non-REM and REM SleepAlternating vigilance states: new insights regarding neuronal networks and mechanisms.Role of the melanin-concentrating hormone neuropeptide in sleep regulation.Neurochemical aspects of sleep regulation with specific focus on slow-wave sleep.Neurochemistry of sleep an overview of animal experimental work.The neuronal network responsible for paradoxical sleep and its dysfunctions causing narcolepsy and rapid eye movement (REM) behavior disorder.Brainstem mechanisms of paradoxical (REM) sleep generation.Paradoxical (REM) sleep genesis by the brainstem is under hypothalamic control.New aspects in the pathophysiology of rapid eye movement sleep behavior disorder: the potential role of glutamate, gamma-aminobutyric acid, and glycine.Rapid eye movement sleep behavior disorder: devising controlled active treatment studies for symptomatic and neuroprotective therapy--a consensus statement from the International Rapid Eye Movement Sleep Behavior Disorder Study Group.Breakdown in REM sleep circuitry underlies REM sleep behavior disorder.Animal models of REM dysfunctions: what they tell us about the cause of narcolepsy and RBD?Neurology and psychiatry: waking up to opportunities of sleep. : State of the art and clinical/research priorities for the next decade.Electrophysiological Evidence That the Retrosplenial Cortex Displays a Strong and Specific Activation Phased with Hippocampal Theta during Paradoxical (REM) Sleep.Not a single but multiple populations of GABAergic neurons control sleep.Selective activation of a few limbic structures during paradoxical (REM) sleep by the claustrum and the supramammillary nucleus: evidence and function.Genetic inactivation of glutamate neurons in the rat sublaterodorsal tegmental nucleus recapitulates REM sleep behaviour disorder.Sleep architecture and homeostasis in mice with partial ablation of melanin-concentrating hormone neurons.Paradoxical (REM) sleep deprivation in mice using the small-platforms-over-water method: polysomnographic analyses and melanin-concentrating hormone and hypocretin/orexin neuronal activation before, during and after deprivation.Paradoxical (REM) sleep deprivation causes a large and rapidly reversible decrease in long-term potentiation, synaptic transmission, glutamate receptor protein levels, and ERK/MAPK activation in the dorsal hippocampus.Evidence that neurons of the sublaterodorsal tegmental nucleus triggering paradoxical (REM) sleep are glutamatergic.Unrelated course of subthalamic nucleus and globus pallidus neuronal activities across vigilance states in the rat.Dopaminergic neurons expressing Fos during waking and paradoxical sleep in the rat.Sleep architecture of the melanin-concentrating hormone receptor 1-knockout mice.Effect of chronic treatment with milnacipran on sleep architecture in rats compared with paroxetine and imipramine.The rat ponto-medullary network responsible for paradoxical sleep onset and maintenance: a combined microinjection and functional neuroanatomical study.The satiety molecule nesfatin-1 is co-expressed with melanin concentrating hormone in tuberal hypothalamic neurons of the rat.
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P50
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հետազոտող
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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Pierre-Hervé Luppi
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P214
P1053
C-4333-2014
P106
P1153
7004127530
P21
P214
P2798
P31
P3829
P496
0000-0002-0503-423X
P734
P7859
lccn-n2004004501