about
Genome-Wide Transcriptional Profiling and Structural Magnetic Resonance Imaging in the Maternal Immune Activation Model of Neurodevelopmental Disorders.Early raise of BDNF in hippocampus suggests induction of posttranscriptional mechanisms by antidepressants.BDNF Val66Met polymorphism and protein levels in amniotic fluidTranscriptomic analyses and leukocyte telomere length measurement in subjects exposed to severe recent stressful life events.Borderline personality disorder and childhood trauma: exploring the affected biological systems and mechanismsStress and inflammation reduce brain-derived neurotrophic factor expression in first-episode psychosis: a pathway to smaller hippocampal volume.Role for the kinase SGK1 in stress, depression, and glucocorticoid effects on hippocampal neurogenesisErbB3 mRNA leukocyte levels as a biomarker for major depressive disorderDepression pathogenesis and treatment: what can we learn from blood mRNA expression?The expression of VGF is reduced in leukocytes of depressed patients and it is restored by effective antidepressant treatment.Antidepressant compounds can be both pro- and anti-inflammatory in human hippocampal cells.Inflammation and neuronal plasticity: a link between childhood trauma and depression pathogenesis.Gene-environment interaction in major depression: focus on experience-dependent biological systems.Glucocorticoids, cytokines and brain abnormalities in depression.Candidate genes expression profile associated with antidepressants response in the GENDEP study: differentiating between baseline 'predictors' and longitudinal 'targets'.Haloperidol and olanzapine mediate metabolic abnormalities through different molecular pathways.Glucocorticoid-related molecular signaling pathways regulating hippocampal neurogenesis.Serum and gene expression profile of cytokines in first-episode psychosis.Transcriptomics in Interferon-α-Treated Patients Identifies Inflammation-, Neuroplasticity- and Oxidative Stress-Related Signatures as Predictors and Correlates of Depression.Ketamine: synaptogenesis, immunomodulation and glycogen synthase kinase-3 as underlying mechanisms of its antidepressant propertiesAbsolute Measurements of Macrophage Migration Inhibitory Factor and Interleukin-1-β mRNA Levels Accurately Predict Treatment Response in Depressed Patients.The human BDNF gene: peripheral gene expression and protein levels as biomarkers for psychiatric disorders.Genetic variations and association.Stress-induced mechanisms in mental illness: A role for glucocorticoid signalling.Effects of mild ozonisation on gene expression and nuclear domains organization in vitro.Cellular and molecular mechanisms of the brain-derived neurotrophic factor in physiological and pathological conditions.Association of brain amyloidosis with pro-inflammatory gut bacterial taxa and peripheral inflammation markers in cognitively impaired elderly.Interleukin-1β: a new regulator of the kynurenine pathway affecting human hippocampal neurogenesis.Antidepressants increase human hippocampal neurogenesis by activating the glucocorticoid receptor.Microbiota and neurodegenerative diseases.Amygdalar MicroRNA-15a Is Essential for Coping with Chronic StressSub-chronic exposure to atomoxetine up-regulates BDNF expression and signalling in the brain of adolescent spontaneously hypertensive rats: comparison with methylphenidate.Long-Term duloxetine treatment normalizes altered brain-derived neurotrophic factor expression in serotonin transporter knockout rats through the modulation of specific neurotrophin isoforms.Reduced function of the serotonin transporter is associated with decreased expression of BDNF in rodents as well as in humans.Reduced peripheral brain-derived neurotrophic factor mRNA levels are normalized by antidepressant treatment.Acute stress responsiveness of the neurotrophin BDNF in the rat hippocampus is modulated by chronic treatment with the antidepressant duloxetine.Integrating 'Omics' Approaches to Prioritize New Pathogenetic Mechanisms for Mental Disorders.Candidate Genes Expression Profile Associated with Antidepressants Response in the GENDEP Study: Differentiating between Baseline ‘Predictors' and Longitudinal ‘Targets'.FoxO1, A2M, and TGF-β1: three novel genes predicting depression in gene X environment interactions are identified using cross-species and cross-tissues transcriptomic and miRNomic analyses.Interferon-Alpha Reduces Human Hippocampal Neurogenesis and Increases Apoptosis via Activation of Distinct STAT1-Dependent Mechanisms.
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P50
description
hulumtuese
@sq
onderzoeker
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researcher
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հետազոտող
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name
Annamaria Cattaneo
@ast
Annamaria Cattaneo
@en
Annamaria Cattaneo
@es
Annamaria Cattaneo
@nl
Annamaria Cattaneo
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type
label
Annamaria Cattaneo
@ast
Annamaria Cattaneo
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Annamaria Cattaneo
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Annamaria Cattaneo
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Annamaria Cattaneo
@sl
prefLabel
Annamaria Cattaneo
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Annamaria Cattaneo
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Annamaria Cattaneo
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Annamaria Cattaneo
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Annamaria Cattaneo
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P1053
K-5847-2016
P106
P1153
55136377000
P21
P31
P3829
P496
0000-0002-9963-848X