Low iron availability modulates the course of Chlamydia pneumoniae infection.
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IFN-gamma-inducible Irga6 mediates host resistance against Chlamydia trachomatis via autophagyThe Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged SwordInnate immune responses to Chlamydia pneumoniae infection: role of TLRs, NLRs, and the inflammasomeMcl-1 is a key regulator of apoptosis resistance in Chlamydia trachomatis-infected cellsTargeted Delivery of Amoxicillin to C. trachomatis by the Transferrin Iron Acquisition PathwayKinematics of intracellular chlamydiae provide evidence for contact-dependent developmentIAP-IAP complexes required for apoptosis resistance of C. trachomatis-infected cells.Gene expression profiles of Chlamydophila pneumoniae during the developmental cycle and iron depletion-mediated persistenceTransgenic, fluorescent Leishmania mexicana allow direct analysis of the proteome of intracellular amastigotes.Wolbachia interferes with ferritin expression and iron metabolism in insectsHaptoglobin and sickle cell polymorphisms and risk of active trachoma in Gambian children.Identification of Chlamydia trachomatis genomic sequences recognized by chlamydial divalent cation-dependent regulator A (DcrA).Increased serum iron levels and infectious complications after liver transplantation.Replication of Neisseria meningitidis within epithelial cells requires TonB-dependent acquisition of host cell iron.Iron and microbial infection.Competitive inhibition of amino acid uptake suppresses chlamydial growth: involvement of the chlamydial amino acid transporter BrnQ.Contribution of the Shigella flexneri Sit, Iuc, and Feo iron acquisition systems to iron acquisition in vitro and in cultured cells.Transcriptional response patterns of Chlamydophila psittaci in different in vitro models of persistent infection.Altered developmental expression of polymorphic membrane proteins in penicillin-stressed Chlamydia trachomatis.Identification and functional analysis of CT069 as a novel transcriptional regulator in ChlamydiaStrategies of Intracellular Pathogens for Obtaining Iron from the EnvironmentChlamydial persistence: beyond the biphasic paradigm.Impact of micro-environmental changes on respiratory tract infections with intracellular bacteria.Examination of an inducible expression system for limiting iron availability during Chlamydia trachomatis infection.Interactions among infections, nutrients and xenobiotics.Influence of iron status on risk of maternal or neonatal infection and on neonatal mortality with an emphasis on developing countries.Genital Chlamydia trachomatis: understanding the roles of innate and adaptive immunity in vaccine research.Iron Homeostasis in Tissues Is Affected during Persistent Chlamydia pneumoniae Infection in Mice.Cholesterol uptake in the mouse aorta increases during Chlamydia pneumoniae infection.Effect of bovine lactoferrin on Chlamydia trachomatis infection and inflammation.Chlamydia trachomatis as the Cause of Infectious Infertility: Acute, Repetitive or Persistent Long-Term Infection?Chlamydia trachomatis remodels stable microtubules to coordinate Golgi stack recruitment to the chlamydial inclusion surface.Targeting eukaryotic Rab proteins: a smart strategy for chlamydial survival and replication.Modulation of host signaling and cellular responses by ChlamydiaAn optimal method of iron starvation of the obligate intracellular pathogen, Chlamydia trachomatis.Analysis of Chlamydia pneumoniae infection in mononuclear cells by reverse transcription-PCR targeted to chlamydial gene transcripts.Reversal of the antichlamydial activity of putative type III secretion inhibitors by iron.NF-kappaB and inhibitor of apoptosis proteins are required for apoptosis resistance of epithelial cells persistently infected with Chlamydophila pneumoniae.Iron enhances endothelial cell activation in response to Cytomegalovirus or Chlamydia pneumoniae infection.Protein expression profiles of Chlamydia pneumoniae in models of persistence versus those of heat shock stress response.
P2860
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P2860
Low iron availability modulates the course of Chlamydia pneumoniae infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 2001
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@en
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@nl
type
label
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@en
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@nl
prefLabel
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@en
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@nl
P2093
P2860
P1476
Low iron availability modulates the course of Chlamydia pneumoniae infection.
@en
P2093
Al-Younes HM
Brinkmann V
Szczepek AJ
P2860
P304
P356
10.1046/J.1462-5822.2001.00125.X
P577
2001-06-01T00:00:00Z