Myelodysplastic syndromes: revisiting the role of the bone marrow microenvironment in disease pathogenesis.
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Bone Marrow Immunity and MyelodysplasiaRevisiting the case for genetically engineered mouse models in human myelodysplastic syndrome researchClinical implications of U2AF1 mutation in patients with myelodysplastic syndrome and its stability during disease progression.Revisiting the hallmarks of cancer.Mitigation of Lethal Radiation Syndrome in Mice by Intramuscular Injection of 3D Cultured Adherent Human Placental Stromal Cells.Increased expression of interferon signaling genes in the bone marrow microenvironment of myelodysplastic syndromes.Defective proliferative potential of MSCs from pediatric myelodysplastic syndrome patients is associated with cell senescence.Gene-expression and in vitro function of mesenchymal stromal cells are affected in juvenile myelomonocytic leukemiaImpaired clearance of apoptotic cells leads to HMGB1 release in the bone marrow of patients with myelodysplastic syndromes and induces TLR4-mediated cytokine production.Navigating the bone marrow niche: translational insights and cancer-driven dysfunction.Effect of IL-7 and IL-15 on T cell phenotype in myelodysplastic syndromes.Dynamics of ASXL1 mutation and other associated genetic alterations during disease progression in patients with primary myelodysplastic syndrome.The hematopoietic stem-cell niche in health and leukemia.Downregulation of MMP1 in MDS-derived mesenchymal stromal cells reduces the capacity to restrict MDS cell proliferation.Regulation of hematopoiesis in endosteal microenvironments.Aging of the hematopoietic stem cells niche.Understanding chronic neutropenia: life is short.Therapy-related myeloid neoplasms: when genetics and environment collide.Phenotypic and Functional Alterations of Hematopoietic Stem and Progenitor Cells in an In Vitro Leukemia-Induced MicroenvironmentMesenchymal stromal cells in myeloid malignancies.Novel therapeutic approach to improve hematopoiesis in low risk MDS by targeting MDSCs with the Fc-engineered CD33 antibody BI 836858Identification of a CD133-CD55- population functions as a fetal common skeletal progenitor.Insufficient stromal support in MDS results from molecular and functional deficits of mesenchymal stromal cells.Inhibition of WNT signaling in the bone marrow niche prevents the development of MDS in the Apcdel/+ MDS mouse model.Aging- and Senescence-associated Changes of Mesenchymal Stromal Cells in Myelodysplastic Syndromes.Senescence of bone marrow mesenchymal stromal cells is accompanied by activation of p53/p21 pathway in myelodysplastic syndromes.Severe hypoxia selects hematopoietic progenitors with stem cell potential from primary Myelodysplastic syndrome bone marrow cell cultures.Clinical significance of hyaluronan levels and its pro-osteogenic effect on mesenchymal stromal cells in myelodysplastic syndromes
P2860
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P2860
Myelodysplastic syndromes: revisiting the role of the bone marrow microenvironment in disease pathogenesis.
description
article científic
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article scientifique
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articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
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artigo científico
@pt-br
artikel ilmiah
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artikull shkencor
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artículo científico
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name
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@en
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@nl
type
label
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@en
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@nl
prefLabel
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@en
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@nl
P2860
P1476
Myelodysplastic syndromes: rev ...... nment in disease pathogenesis.
@en
P2093
Marc H G P Raaijmakers
P2860
P2888
P356
10.1007/S12185-011-1001-X
P577
2012-01-06T00:00:00Z