Aconitase post-translational modification as a key in linkage between Krebs cycle, iron homeostasis, redox signaling, and metabolism of reactive oxygen species.
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Mitochondrial Lon protease in human disease and aging: Including an etiologic classification of Lon-related diseases and disordersROS homeostasis and metabolism: a critical liaison for cancer therapyPlasma metabolomics reveals a diagnostic metabolic fingerprint for mitochondrial aconitase (ACO2) deficiencyFumarate metabolism and ATP production in Pseudomonas fluorescens exposed to nitrosative stress.Differential cysteine labeling and global label-free proteomics reveals an altered metabolic state in skeletal muscle aging.Bioenergetic programming of macrophages by the apolipoprotein A-I mimetic peptide 4FA novel mutation in nuclear prelamin a recognition factor-like causes diffuse pulmonary arteriovenous malformationsAccumulation of Palmitoylcarnitine and Its Effect on Pro-Inflammatory Pathways and Calcium Influx in Prostate Cancer.Molecular Evolution and Expression Divergence of the Aconitase (ACO) Gene Family in Land Plants.Thiol switches in mitochondria: operation and physiological relevance.Metabolic control of signalling pathways and metabolic auto-regulation.Localisation and protein-protein interactions of the Helicobacter pylori taxis sensor TlpD and their connection to metabolic functionsMitochondrial iron and energetic dysfunction distinguish fibroblasts and induced neurons from pantothenate kinase-associated neurodegeneration patients.Protein phosphatase 2A (PP2A) regulatory subunit B'γ interacts with cytoplasmic ACONITASE 3 and modulates the abundance of AOX1A and AOX1D in Arabidopsis thaliana.Running-wheel activity delays mitochondrial respiratory flux decline in aging mouse muscle via a post-transcriptional mechanism.Alpha-ketoglutarate enhances freeze-thaw tolerance and prevents carbohydrate-induced cell death of the yeast Saccharomyces cerevisiae.Complete Mitochondrial Complex I Deficiency Induces an Up-Regulation of Respiratory Fluxes That Is Abolished by Traces of Functional Complex I.Redox Paradox: A novel approach to therapeutics-resistant cancer.Mitochondrial toxicity of tobacco smoke and air pollution.
P2860
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P2860
Aconitase post-translational modification as a key in linkage between Krebs cycle, iron homeostasis, redox signaling, and metabolism of reactive oxygen species.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh
2013年學術文章
@zh-hant
name
Aconitase post-translational m ...... sm of reactive oxygen species.
@en
type
label
Aconitase post-translational m ...... sm of reactive oxygen species.
@en
prefLabel
Aconitase post-translational m ...... sm of reactive oxygen species.
@en
P2860
P1433
P1476
Aconitase post-translational m ...... ism of reactive oxygen species
@en
P2093
Oleh V Lushchak
Volodymyr I Lushchak
P2860
P356
10.1179/1351000213Y.0000000073
P577
2013-11-22T00:00:00Z