Pathological mitochondrial copper overload in livers of Wilson's disease patients and related animal models.
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Angiotensin II type 1 receptor blockers increase tolerance of cells to copper and cisplatin.Optical coherence tomography and electrophysiology of retinal and visual pathways in Wilson's diseaseSphingolipids and mitochondrial function, lessons learned from yeastBioavailable Trace Metals in Neurological Diseases.A proteomic-based investigation of potential copper-responsive biomarkers: Proteins, conceptual networks, and metabolic pathways featuring Penicillium janthinellum from a heavy metal-polluted ecological nicheMethanobactin reverses acute liver failure in a rat model of Wilson disease.The plant decapeptide OSIP108 can alleviate mitochondrial dysfunction induced by cisplatin in human cells.The mitochondrion: a central architect of copper homeostasis.Exchangeable copper: a reflection of the neurological severity in Wilson's disease.
P2860
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P2860
Pathological mitochondrial copper overload in livers of Wilson's disease patients and related animal models.
description
article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
Pathological mitochondrial cop ...... nts and related animal models.
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type
label
Pathological mitochondrial cop ...... nts and related animal models.
@en
prefLabel
Pathological mitochondrial cop ...... nts and related animal models.
@en
P2860
P356
P1476
Pathological mitochondrial cop ...... ents and related animal models
@en
P2093
Josef Lichtmannegger
P2860
P356
10.1111/NYAS.12347
P407
P50
P577
2014-02-12T00:00:00Z