Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes - sprookjes - yvandenbroek - TriplyDB

Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes

Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes

http://www.wikidata.org/entity/Q38332511

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Diabetic complications within the context of aging: Nicotinamide adenine dinucleotide redox, insulin C-peptide, sirtuin 1-liver kinase B1-adenosine monophosphate-activated protein kinase positive feedback and forkhead box O3 Histological changes of kidney in diabetic nephropathy Development of the Mammalian Kidney C3a receptor antagonist ameliorates inflammatory and fibrotic signals in type 2 diabetic nephropathy by suppressing the activation of TGF-β/smad3 and IKBα pathway Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.Apelin promotes diabetic nephropathy by inducing podocyte dysfunction via inhibiting proteasome activities.Stem Cell-Derived, microRNA-Carrying Extracellular Vesicles: A Novel Approach to Interfering with Mesangial Cell Collagen Production in a Hyperglycaemic Setting.Xiaokeping mixture inhibits diabetic nephropathy in streptozotocin-induced rats through blocking TGF-β1/Smad7 signaling Coronary Artery Disease Is a Predictor of Progression to Dialysis in Patients With Chronic Kidney Disease, Type 2 Diabetes Mellitus, and Anemia: An Analysis of the Trial to Reduce Cardiovascular Events With Aranesp Therapy (TREAT).Early protective role of MST1 knockdown in response to experimental diabetic nephropathy Silencing of Histone Deacetylase 9 Expression in Podocytes Attenuates Kidney Injury in Diabetic Nephropathy.Nicousamide protects kidney podocyte by inhibiting the TGFβ receptor II phosphorylation and AGE-RAGE signaling Soluble epoxide hydrolase in podocytes is a significant contributor to renal function under hyperglycemia.Targeted functional investigations guided by integrative proteome network analysis revealed significant perturbations of renal tubular cell functions induced by high glucose.Effects of ellagic acid pretreatment on renal functions disturbances induced by global cerebral ischemic-reperfusion in rat.Vascular endothelium in diabetes.Classification and Differential Diagnosis of Diabetic Nephropathy.Protection of CTGF Antibody Against Diabetic Nephropathy in Mice Via Reducing Glomerular β-Catenin Expression and Podocyte Epithelial-Mesenchymal Transition.Polydatin impairs mitochondria fitness and ameliorates podocyte injury by suppressing Drp1 expression.Research Progress on Mechanism of Podocyte Depletion in Diabetic Nephropathy.Diabetic Nephropathy: a Tangled Web to Unweave.Murine glomerular transcriptome links endothelial cell-specific molecule-1 deficiency with susceptibility to diabetic nephropathy.Lysyl oxidase‑like 2 is expressed in kidney tissue and is associated with the progression of tubulointerstitial fibrosis.MDM2 is implicated in high-glucose-induced podocyte mitotic catastrophe via Notch1 signalling.Apelin/APJ system: A novel potential therapy target for kidney disease.Podocyte-derived microparticles promote proximal tubule fibrotic signaling via p38 MAPK and CD36.LY333531, a PKCβ inhibitor, attenuates glomerular endothelial cell apoptosis in the early stage of mouse diabetic nephropathy via down-regulating swiprosin-1.Endocan and albuminuria in type 2 diabetes mellitus.Acute Kidney Injury and Progression of Diabetic Kidney Disease.Treatment with irbesatan may improve slit diaphragm alterations in rats with adriamycin-induced nephropathy.Protective effects of leflunomide on renal lesions in a rat model if diabetic nephropathy.AMPKα, hs-CRP and FcγR in diabetic nephropathy and drug intervention.Revealing Synergistic Mechanism of Multiple Components in Gandi Capsule for Diabetic Nephropathy Therapeutics by Network Pharmacology.NaoXinTong Capsules inhibit the development of diabetic nephropathy in db/db mice.Stem/Stromal Cells for Treatment of Kidney Injuries With Focus on Preclinical Models.

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Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes

http://www.wikidata.org/entity/Q38332511

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2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

@zh-sg

2014年學術文章

@yue

2014年學術文章

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2014年學術文章

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name

Cell biology of diabetic nephr ...... terstitial cells and podocytes

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Cell biology of diabetic nephr ...... terstitial cells and podocytes

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Cell biology of diabetic nephr ...... terstitial cells and podocytes

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Journal of Diabetes Investigation

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Cell biology of diabetic nephr ...... terstitial cells and podocytes

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Koutaro Yokote

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Minoru Takemoto

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Yoshiro Maezawa

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P2860

Nck adaptor proteins link nephrin to the actin cytoskeleton of kidney podocytes

Angiopoietin 2 is a partial agonist/antagonist of Tie2 signaling in the endothelium

Angiopoietin-2, a natural antagonist for Tie2 that disrupts in vivo angiogenesis

Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome

Angiopoietin-1 is essential in mouse vasculature during development and in response to injury.

mTOR signaling in growth control and disease

Cell biology and pathology of podocytes

Diabetic nephropathy and extracellular matrix

Expression of podocyte-associated molecules in acquired human kidney diseases

Proteinuria and the expression of the podocyte slit diaphragm protein, nephrin, in diabetic nephropathy: effects of angiotensin converting enzyme inhibition

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10.1111/JDI.12255

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