Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications. - sprookjes - yvandenbroek - TriplyDB

Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

http://www.wikidata.org/entity/Q38756562

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When Insult Is Added to Injury: Cross Talk between ILCs and Intestinal Epithelium in IBD IL-33-induced alternatively activated macrophage attenuates the development of TNBS-induced colitis.Intestinal Autophagy and Its Pharmacological Control in Inflammatory Bowel Disease.Biomarkers in Search of Precision Medicine in IBD.Advances in the understanding of mitochondrial DNA as a pathogenic factor in inflammatory diseases.Stromal and immune cells in gut fibrosis: the myofibroblast and the scarface.Comparison of acute kidney injury of different aetiology reveals in-common mechanisms of tissue damage.Smad nuclear interacting protein 1 (SNIP1) inhibits intestinal inflammation through regulation of epithelial barrier function.Association between serum S100A9 levels and liver necroinflammation in chronic hepatitis B.S100A8/A9 in Inflammation.Damage-associated molecular patterns in inflammatory bowel disease: From biomarkers to therapeutic targets Regulation of S100A8 Stability by RNF5 in Intestinal Epithelial Cells Determines Intestinal Inflammation and Severity of Colitis

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P2860

Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

http://www.wikidata.org/entity/Q38756562

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

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2016年论文

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name

Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

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Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

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Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

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Mucosal Immunology

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Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.

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A G Rossi

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G-T Ho

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J Satsangi

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R K Boyapati

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P2860

Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans

Identification of human S100A9 as a novel target for treatment of autoimmune disease via binding to quinoline-3-carboxamides

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Increasing Incidence and Prevalence of the Inflammatory Bowel Diseases With Time, Based on Systematic Review

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase

Induction of immunological tolerance by apoptotic cells requires caspase-dependent oxidation of high-mobility group box-1 protein

Loss of S100A9 (MRP14) results in reduced interleukin-8-induced CD11b surface expression, a polarized microfilament system, and diminished responsiveness to chemoattractants in vitro

A novel pathway combining calreticulin exposure and ATP secretion in immunogenic cancer cell death

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567-582

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10.1038/MI.2016.14

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3

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