Upregulation of nuclear factor-κB activity mediates CYP24 expression and reactive oxygen species production in indoxyl sulfate-induced chronic kidney disease.

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Role of Vitamin D in Uremic Vascular Calcification.Vitamin D alleviates lead induced renal and testicular injuries by immunomodulatory and antioxidant mechanisms in rats.

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Upregulation of nuclear factor-κB activity mediates CYP24 expression and reactive oxygen species production in indoxyl sulfate-induced chronic kidney disease.

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Upregulation of nuclear factor ...... nduced chronic kidney disease.

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Upregulation of nuclear factor ...... nduced chronic kidney disease.

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Upregulation of nuclear factor ...... nduced chronic kidney disease.

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Lili Wang

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Yongning Gao

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P2860

N-acetylcysteine for sepsis and systemic inflammatory response in adults

p38 MAPK phosphorylation and NF-kappa B activation in human crescentic glomerulonephritis

Tumor necrosis factor-alpha-induced IKK phosphorylation of NF-kappaB p65 on serine 536 is mediated through the TRAF2, TRAF5, and TAK1 signaling pathway

Crosstalk between the unfolded protein response and NF-κB-mediated inflammation in the progression of chronic kidney disease

TNF-alpha inhibition reduces renal injury in DOCA-salt hypertensive rats.

Regulation of redox glutathione levels and gene transcription in lung inflammation: therapeutic approaches.

Clove extract inhibits tumor growth and promotes cell cycle arrest and apoptosis.

Krüppel-like factor 5 mediates proinflammatory cytokine expression in lipopolysaccharide-induced acute lung injury through upregulation of nuclear factor-κB phosphorylation in vitro and in vivo

Indoxyl sulfate, a uremic toxin, downregulates renal expression of Nrf2 through activation of NF-κB.

Renal damage associated with proteinuria.

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Upregulation of nuclear factor-κB activity mediates CYP24 expression and reactive oxygen species production in indoxyl sulfate-induced chronic kidney disease.

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P2860

P2860

Upregulation of nuclear factor-κB activity mediates CYP24 expression and reactive oxygen species production in indoxyl sulfate-induced chronic kidney disease.

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