about
High frequency of gastrointestinal manifestations in myotonic dystrophy type 1 and type 2.Hybrid splicing minigene and antisense oligonucleotides as efficient tools to determine functional protein/RNA interactions.RNA-binding proteins in neurodegeneration: mechanisms in aggregate.MBNL expression in autoregulatory feedback loops.Sarcolemmal excitability in the myotonic dystrophies.Selective alkylation of T-T mismatched DNA using vinyldiaminotriazine-acridine conjugate.Detection of expanded RNA repeats using thermostable group II intron reverse transcriptase.Gene Editing and Gene-Based Therapeutics for Cardiomyopathies.Abnormalities in Skeletal Muscle Myogenesis, Growth, and Regeneration in Myotonic Dystrophy.miR-23b and miR-218 silencing increase Muscleblind-like expression and alleviate myotonic dystrophy phenotypes in mammalian models.Alternative splicing analysis in human monocytes and macrophages reveals MBNL1 as major regulator
P2860
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P2860
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Myotonic dystrophy: approach to therapy.
@en
type
label
Myotonic dystrophy: approach to therapy.
@en
prefLabel
Myotonic dystrophy: approach to therapy.
@en
P2093
P2860
P1476
Myotonic dystrophy: approach to therapy.
@en
P2093
Charles A Thornton
Ellie M Carrell
P2860
P304
P356
10.1016/J.GDE.2017.03.007
P577
2017-04-01T00:00:00Z