Oct4 mediates tumor initiating properties in oral squamous cell carcinomas through the regulation of epithelial-mesenchymal transition. - sprookjes - yvandenbroek - TriplyDB

Oct4 mediates tumor initiating properties in oral squamous cell carcinomas through the regulation of epithelial-mesenchymal transition.

Oct4 mediates tumor initiating properties in oral squamous cell carcinomas through the regulation of epithelial-mesenchymal transition.

http://www.wikidata.org/entity/Q39029342

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Tumorsphere as an effective in vitro platform for screening anti-cancer stem cell drugs Potential Gene Interactions in the Cell Cycles of Gametes, Zygotes, Embryonic Stem Cells and the Development of Cancer OCT4 expression and vasculogenic mimicry formation positively correlate with poor prognosis in human breast cancer.Transglutaminase Is Required for Epidermal Squamous Cell Carcinoma Stem Cell Survival.Induction of artificial cancer stem cells from tongue cancer cells by defined reprogramming factors.Simultaneous knockdown of uPA and MMP9 can reduce breast cancer progression by increasing cell-cell adhesion and modulating EMT genes.NANOG regulates epithelial-mesenchymal transition and chemoresistance through activation of the STAT3 pathway in epithelial ovarian cancer.miR-367 promotes proliferation and stem-like traits in medulloblastoma cells.Anoikis-resistant subpopulations of human osteosarcoma display significant chemoresistance and are sensitive to targeted epigenetic therapies predicted by expression profiling Cancer Stem Cells in Oral Cavity Squamous Cell Carcinoma: A Review.Effect of HPV on tumor expression levels of the most commonly used markers in HNSCC.Acquisition cancer stemness, mesenchymal transdifferentiation, and chemoresistance properties by chronic exposure of oral epithelial cells to arecoline.Cancer stem cells in moderately differentiated oral tongue squamous cell carcinoma.Transcriptional factor OCT4 promotes esophageal cancer metastasis by inducing epithelial-mesenchymal transition through VEGF-C/VEGFR-3 signaling pathway.Concurrent expression of Oct4 and Nanog maintains mesenchymal stem-like property of human dental pulp cells.Targeting LncRNA HOTAIR suppresses cancer stemness and metastasis in oral carcinomas stem cells through modulation of EMT.Plasminogen activator inhibitor-1 as regulator of tumor-initiating cell properties in head and neck cancers.GLI3 knockdown decreases stemness, cell proliferation and invasion in oral squamous cell carcinoma Exposure to nicotine-derived nitrosamine ketone and arecoline synergistically facilitates tumor aggressiveness via overexpression of epidermal growth factor receptor and its downstream signaling in head and neck squamous cell carcinoma miR-1246 Targets CCNG2 to Enhance Cancer Stemness and Chemoresistance in Oral Carcinomas

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Oct4 mediates tumor initiating properties in oral squamous cell carcinomas through the regulation of epithelial-mesenchymal transition.

http://www.wikidata.org/entity/Q39029342

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年论文

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2014年论文

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name

Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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type

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Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

@en

Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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Oct4 mediates tumor initiating ...... helial-mesenchymal transition.

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Cheng-Chia Yu

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Chuan-Hang Yu

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Fang-Wei Hu

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Lo-Lin Tsai

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Shiuan-Shinn Lee

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Yu-Chao Chang

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P2860

Direct regulation of TWIST by HIF-1alpha promotes metastasis

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The epithelial-mesenchymal transition generates cells with properties of stem cells

Cancer statistics, 2013

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Reprogramming of human somatic cells to pluripotency with defined factors

A protein interaction network for pluripotency of embryonic stem cells

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Oct-4 expression maintained cancer stem-like properties in lung cancer-derived CD133-positive cells

Kaempferol reduces matrix metalloproteinase-2 expression by down-regulating ERK1/2 and the activator protein-1 signaling pathways in oral cancer cells

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