The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
about
HRD1 prevents apoptosis in renal tubular epithelial cells by mediating eIF2α ubiquitylation and degradation.Lysyl oxidases expression and histopathological changes of the diabetic rat nephron.Fatty acid receptor modulator PBI-4050 inhibits kidney fibrosis and improves glycemic control.Renal tubular epithelial cells: the neglected mediator of tubulointerstitial fibrosis after injury
P2860
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@en
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@nl
type
label
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@en
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@nl
prefLabel
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@en
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@nl
P2093
P2860
P1476
The Role of Endoplasmic Reticulum Stress in Diabetic Nephropathy.
@en
P2093
John Cijiang He
Niansong Wang
P2860
P2888
P356
10.1007/S11892-017-0842-Y
P50
P577
2017-03-01T00:00:00Z