Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
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Clinical implications of cardiac hyperpolarized magnetic resonance imagingThe pigeon heart 5'-nucleotidase responsible for ischaemia-induced adenosine formationLocation and properties of two isoenzymes of cardiac adenylate kinaseMetabolic dependence of glycolytic enzyme binding in rat and sheep heartExpression of skeletal muscle sodium channel (Nav1.4) or connexin32 prevents reperfusion arrhythmias in murine heartInhibition of p38 MAPK alpha/beta reduces ischemic injury and does not block protective effects of preconditioning.Effect of pH and counterions on the encapsulation properties of xenon in water-soluble cryptophanes.Measuring intracellular pH in the heart using hyperpolarized carbon dioxide and bicarbonate: a 13C and 31P magnetic resonance spectroscopy studyCombined troponin I Ser-150 and Ser-23/24 phosphorylation sustains thin filament Ca(2+) sensitivity and accelerates deactivation in an acidic environment.Ion transport and energetics during cell death and protection.pH-dependent modulation of voltage gating in connexin45 homotypic and connexin45/connexin43 heterotypic gap junctionsActivity of the Na+/H+ exchanger contributes to cardiac damage following ischaemia and reperfusion.A structural basis for the unequal sensitivity of the major cardiac and liver gap junctions to intracellular acidification: the carboxyl tail lengthExperimental generation and computational modeling of intracellular pH gradients in cardiac myocytes.Syntaxin-1A actions on sulfonylurea receptor 2A can block acidic pH-induced cardiac K(ATP) channel activation.A dynamic model of excitation-contraction coupling during acidosis in cardiac ventricular myocytes.Intracellular pH during "chemical hypoxia" in cultured rat hepatocytes. Protection by intracellular acidosis against the onset of cell deathOxygen at physiological concentrations. A potential, paradoxical mediator of reperfusion injury to mitochondria induced by phosphateProton-sensing Ca2+ binding domains regulate the cardiac Na+/Ca2+ exchanger.Influence of pH on Ca²⁺ current and its control of electrical and Ca²⁺ signaling in ventricular myocytes.pH-Dependence of extrinsic and intrinsic H(+)-ion mobility in the rat ventricular myocyte, investigated using flash photolysis of a caged-H(+) compound.pH-dependent modulation of intracellular free magnesium ions with ion-selective electrodes in papillary muscle of guinea pig.Magnetic resonance for the anaesthetist. Part I: Physical principles, applications, safety aspects.SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmicEffect of skeletal muscle Na(+) channel delivered via a cell platform on cardiac conduction and arrhythmia induction.Protection of the ischemic myocardium during the reperfusion: between hope and reality.Mechanisms of cytoplasmic pH regulation in hypoxic maize root tips and its role in survival under hypoxia.Hyperpolarized 13C allows a direct measure of flux through a single enzyme-catalyzed step by NMR.Mitochondrial regulation of phosphocreatine/inorganic phosphate ratios in exercising human muscle: a gated 31P NMR studyNoninvasive, nondestructive approaches to cell bioenergetics.A critical assessment of methods of measuring metabolite concentrations by NMR spectroscopy.Complementarity of magnetic resonance spectroscopy, positron emission tomography and single photon emission tomography for the in vivo investigation of human cardiac metabolism and neurotransmission.pH-Regulated Na(+) influx into the mammalian ventricular myocyte: the relative role of Na(+)-H(+) exchange and Na(+)-HCO Co-transport.Noninvasive tissue characterization of myocardium by topical 1H-and 31P-nuclear magnetic resonance spectroscopy.Ca2+/calmodulin-dependent protein kinase: a key component in the contractile recovery from acidosis.Potential and limitations of nuclear magnetic resonance for the cardiologist.Study of ischemic myocardial buffering capacity in perfused rat heart through rapidly interleaved 1H and 31P MRS measurements.Inhibition of carbohydrate oxidation during the first minute of reperfusion after brief ischemia: NMR detection of hyperpolarized 13CO2 and H13CO3-.Modulation of the cardiac sodium/bicarbonate cotransporter by the renin angiotensin aldosterone system: pathophysiological consequences.Cytoplasmic acidosis as a determinant of flooding intolerance in plants
P2860
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P2860
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
description
1979 nî lūn-bûn
@nan
1979年の論文
@ja
1979年学术文章
@wuu
1979年学术文章
@zh-cn
1979年学术文章
@zh-hans
1979年学术文章
@zh-my
1979年学术文章
@zh-sg
1979年學術文章
@yue
1979年學術文章
@zh
1979年學術文章
@zh-hant
name
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
@en
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance.
@nl
type
label
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
@en
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance.
@nl
prefLabel
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
@en
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance.
@nl
P2093
P2860
P356
P1433
P1476
Studies of acidosis in the ischaemic heart by phosphorus nuclear magnetic resonance
@en
P2093
P2860
P304
P356
10.1042/BJ1840547
P407
P577
1979-12-01T00:00:00Z