Chemokine gene expression in astrocytes of Borna disease virus-infected rats and mice in the absence of inflammation.
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Functional expression of CCR2 by human fetal astrocytesImmunological and PCR analyses for Borna disease virus in psychiatric patients and blood donors in JapanConstitutive activation of the transcription factor NF-kappaB results in impaired borna disease virus replication.Alpha/beta interferon promotes transcription and inhibits replication of borna disease virus in persistently infected cells.Borna disease virus and human disease.Cerebral expression of interleukin-12 induces neurological disease via differential pathways and recruits antigen-specific T cells in virus-infected mice.Astrocytes play a key role in activation of microglia by persistent Borna disease virus infection.Interactions between TLR7 and TLR9 agonists and receptors regulate innate immune responses by astrocytes and microglia.Astrocytes: Integrative Regulators of Neuroinflammation in Stroke and Other Neurological Diseases.Animal models of CNS viral disease: examples from borna disease virus models.Interferon-independent, human immunodeficiency virus type 1 gp120-mediated induction of CXCL10/IP-10 gene expression by astrocytes in vivo and in vitro.Identification of genes involved in the host response to neurovirulent alphavirus infection.Borna disease virus accelerates inflammation and disease associated with transgenic expression of interleukin-12 in the central nervous system.Selective virus resistance conferred by expression of Borna disease virus nucleocapsid components.CXCL10 production from cytomegalovirus-stimulated microglia is regulated by both human and viral interleukin-10.Borna disease virus multiplication in mouse organotypic slice cultures is site-specifically inhibited by gamma interferon but not by interleukin-12.Borna disease virus replication in organotypic hippocampal slice cultures from rats results in selective damage of dentate granule cells.Electrophysiological alterations and upregulation of ATP receptors in retinal glial Müller cells from rats infected with the Borna disease virus.Morphometric analysis of the retina from horses infected with the Borna disease virus.Borna Disease Virus
P2860
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P2860
Chemokine gene expression in astrocytes of Borna disease virus-infected rats and mice in the absence of inflammation.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Chemokine gene expression in a ...... n the absence of inflammation.
@en
Chemokine gene expression in a ...... n the absence of inflammation.
@nl
type
label
Chemokine gene expression in a ...... n the absence of inflammation.
@en
Chemokine gene expression in a ...... n the absence of inflammation.
@nl
prefLabel
Chemokine gene expression in a ...... n the absence of inflammation.
@en
Chemokine gene expression in a ...... n the absence of inflammation.
@nl
P2093
P2860
P1433
P1476
Chemokine gene expression in a ...... n the absence of inflammation.
@en
P2093
A Pagenstecher
J Hausmann
P Staeheli
S Schneckenburger
W Hallensleben
P2860
P304
P356
10.1128/JVI.74.19.9267-9280.2000
P407
P577
2000-10-01T00:00:00Z