Interactions between the juvenile Batten disease gene, CLN3, and the Notch and JNK signalling pathways.
about
Neuronal ceroid lipofuscinosis protein CLN3 interacts with motor proteins and modifies location of late endosomal compartmentsThe FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomesInteractions of the proteins of neuronal ceroid lipofuscinosis: clues to functionCerebellar defects in a mouse model of juvenile neuronal ceroid lipofuscinosis.Genetic studies in Drosophila and humans support a model for the concerted function of CISD2, PPT1 and CLN3 in disease.Interaction between Sdo1p and Btn1p in the Saccharomyces cerevisiae model for Batten disease.Identifying cellular pathways modulated by Drosophila palmitoyl-protein thioesterase 1 function.The Batten disease gene CLN3 is required for the response to oxidative stress.Wnt/β-catenin signaling integrates patterning and metabolism of the insect growth zone.Royal Decree: Gene Expression in Trans-Generationally Immune Primed Bumblebee Workers Mimics a Primary Immune ResponseRecent advances in using Drosophila to model neurodegenerative diseases.Mutations in palmitoyl-protein thioesterase 1 alter exocytosis and endocytosis at synapses in Drosophila larvae.A saposin deficiency model in Drosophila: Lysosomal storage, progressive neurodegeneration and sensory physiological declineInvertebrate models of lysosomal storage disease: what have we learned so far?Odd-skipped labels a group of distinct neurons associated with the mushroom body and optic lobe in the adult Drosophila brain.Diminished MTORC1-Dependent JNK Activation Underlies the Neurodevelopmental Defects Associated with Lysosomal DysfunctionModification of the Drosophila model of in vivo Tau toxicity reveals protective phosphorylation by GSK3β.A central role for TOR signalling in a yeast model for juvenile CLN3 diseaseProteomic mapping of differentially vulnerable pre-synaptic populations identifies regulators of neuronal stability in vivoin vivo localization of the neuronal ceroid lipofuscinosis proteins, CLN3 and CLN7, at endogenous expression levelsDefective synaptic transmission causes disease signs in a mouse model of juvenile neuronal ceroid lipofuscinosis.
P2860
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P2860
Interactions between the juvenile Batten disease gene, CLN3, and the Notch and JNK signalling pathways.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Interactions between the juven ...... h and JNK signalling pathways.
@en
Interactions between the juven ...... h and JNK signalling pathways.
@nl
type
label
Interactions between the juven ...... h and JNK signalling pathways.
@en
Interactions between the juven ...... h and JNK signalling pathways.
@nl
prefLabel
Interactions between the juven ...... h and JNK signalling pathways.
@en
Interactions between the juven ...... h and JNK signalling pathways.
@nl
P2860
P356
P1476
Interactions between the juven ...... ch and JNK signalling pathways
@en
P2093
Megan B O'Hare
Valérie Vivancos
P2860
P304
P356
10.1093/HMG/DDN396
P577
2008-11-21T00:00:00Z