Human immunodeficiency virus type 1 DNA synthesis, integration, and efficient viral replication in growth-arrested T cells.
about
HIV-1 nuclear import: matrix protein is back on center stage, this time together with VprNuclear localization signal of HIV-1 as a novel target for therapeutic interventionPrimitive hematopoietic cells resist HIV-1 infection via p21Inhibition of human immunodeficiency virus type 1 transcription by chemical cyclin-dependent kinase inhibitorsProgression to the G1b phase of the cell cycle is required for completion of human immunodeficiency virus type 1 reverse transcription in T cells.Kinetics of human immunodeficiency virus type 1 (HIV) DNA integration in acutely infected cells as determined using a novel assay for detection of integrated HIV DNA.Transduction of interphase cells by avian sarcoma virus.Immediate activation fails to rescue efficient human immunodeficiency virus replication in quiescent CD4+ T cells.Extracellular Vpr protein increases cellular permissiveness to human immunodeficiency virus replication and reactivates virus from latency.Interaction of nuclear protein p140 with human immunodeficiency virus type 1 TAR RNA in mitogen-activated primary human T lymphocytes.A hard way to the nucleusFormation of simian immunodeficiency virus long terminal repeat circles in resting T cells requires both T cell receptor- and IL-2-dependent activationInhibition of early and late events of the HIV-1 replication cycle by cytoplasmic Fab intrabodies against the matrix protein, p17Efficient gene transfer into nondividing cells by adeno-associated virus-based vectors.Integrase mutants of human immunodeficiency virus type 1 with a specific defect in integration.Transduction of nondividing cells using pseudotyped defective high-titer HIV type 1 particles.Minimal requirement for a lentivirus vector based on human immunodeficiency virus type 1.Diminished human immunodeficiency virus type 1 reverse transcription and nuclear transport in primary macrophages arrested in early G(1) phase of the cell cycleNonproductive human immunodeficiency virus type 1 infection in nucleoside-treated G0 lymphocytes.Effect of cell cycle arrest on the activity of nucleoside analogues against human immunodeficiency virus type 1.Passage through mitosis is required for oncoretroviruses but not for the human immunodeficiency virus.Reverse transcription complex: the key player of the early phase of HIV replication.Productive HIV-1 infection of macrophages restricted to the cell fraction with proliferative capacity.
P2860
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P2860
Human immunodeficiency virus type 1 DNA synthesis, integration, and efficient viral replication in growth-arrested T cells.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Human immunodeficiency virus t ...... on in growth-arrested T cells.
@en
type
label
Human immunodeficiency virus t ...... on in growth-arrested T cells.
@en
prefLabel
Human immunodeficiency virus t ...... on in growth-arrested T cells.
@en
P2093
P2860
P1433
P1476
Human immunodeficiency virus t ...... ion in growth-arrested T cells
@en
P2093
P2860
P304
P407
P577
1993-07-01T00:00:00Z