In vivo Cigarette Smoke Exposure Decreases CCL20, SLPI, and BD-1 Secretion by Human Primary Nasal Epithelial Cells.
about
SIRT1/FoxO3 axis alteration leads to aberrant immune responses in bronchial epithelial cells.Airway surface liquid from smokers promotes bacterial growth and biofilm formation via iron-lactoferrin imbalance.Tobacco Smoke Induces and Alters Immune Responses in the Lung Triggering Inflammation, Allergy, Asthma and Other Lung Diseases: A Mechanistic Review.
P2860
In vivo Cigarette Smoke Exposure Decreases CCL20, SLPI, and BD-1 Secretion by Human Primary Nasal Epithelial Cells.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
@zh-hant
2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
In vivo Cigarette Smoke Exposu ...... rimary Nasal Epithelial Cells.
@en
type
label
In vivo Cigarette Smoke Exposu ...... rimary Nasal Epithelial Cells.
@en
prefLabel
In vivo Cigarette Smoke Exposu ...... rimary Nasal Epithelial Cells.
@en
P2093
P2860
P356
P1476
In vivo Cigarette Smoke Exposu ...... rimary Nasal Epithelial Cells.
@en
P2093
Benoit J Gosselin
James Jukosky
Leah Foley
Mardi A Crane-Godreau
Steven Fiering
Tenzin Dechen
P2860
P356
10.3389/FPSYT.2015.00185
P577
2015-01-01T00:00:00Z