Depolarization and decreased surface expression of K+ channels contribute to NSAID-inhibition of intestinal restitution. - sprookjes - yvandenbroek - TriplyDB

Depolarization and decreased surface expression of K+ channels contribute to NSAID-inhibition of intestinal restitution.

Depolarization and decreased surface expression of K+ channels contribute to NSAID-inhibition of intestinal restitution.

http://www.wikidata.org/entity/Q40133487

about

Potassium Channelopathies and Gastrointestinal Ulceration Nonsteroidal anti-inflammatory drugs and the risk for anastomotic failure: a report from Washington State's Surgical Care and Outcomes Assessment Program (SCOAP).Inhibition of Kv channel expression by NSAIDs depolarizes membrane potential and inhibits cell migration by disrupting calpain signaling Ketorolac Use and Postoperative Complications in Gastrointestinal Surgery.Effects of analgesics and antidepressants on TREK-2 and TRESK currents Insights into the effects of diclofenac and other non-steroidal anti-inflammatory agents on ion channels.Suppression of calpain expression by NSAIDs is associated with inhibition of cell migration in rat duodenum.Expression of pleiotrophin, an important regulator of cell migration, is inhibited in intestinal epithelial cells by treatment with non-steroidal anti-inflammatory drugs.Non-steroidal anti-inflammatory drugs inhibit calpain activity and membrane localization of calpain 2 protease.Putting the brakes on vascular smooth muscle cell migration.Pharmacological activation of epidermal growth factor receptor signaling inhibits colitis-associated cancer in mice.

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P2860

Depolarization and decreased surface expression of K+ channels contribute to NSAID-inhibition of intestinal restitution.

http://www.wikidata.org/entity/Q40133487

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2007 nî lūn-bûn

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Depolarization and decreased s ...... ion of intestinal restitution.

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Depolarization and decreased s ...... ion of intestinal restitution.

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Depolarization and decreased s ...... ion of intestinal restitution.

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Biochemical Pharmacology

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Depolarization and decreased s ...... ion of intestinal restitution.

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A McCoy

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P2860

Subunit composition determines Kv1 potassium channel surface expression

Inhibition of store-operated calcium entry contributes to the anti-proliferative effect of non-steroidal anti-inflammatory drugs in human colon cancer cells

NSAIDs induce both necrosis and apoptosis in guinea pig gastric mucosal cells in primary culture

Polyamine depletion stabilizes p53 resulting in inhibition of normal intestinal epithelial cell proliferation

Etoricoxib (MK-0663): preclinical profile and comparison with other agents that selectively inhibit cyclooxygenase-2.

Mechanisms and modulation of intestinal epithelial repair.

Effects of the atrial antiarrhythmic drug AVE0118 on cardiac ion channels.

Uncoupling of intestinal mitochondrial oxidative phosphorylation and inhibition of cyclooxygenase are required for the development of NSAID-enteropathy in the rat.

Identification of a trafficking determinant localized to the Kv1 potassium channel pore

Where is the evidence that cyclooxygenase inhibition is the primary cause of nonsteroidal anti-inflammatory drug (NSAID)-induced gastrointestinal injury? Topical injury revisited.

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