Expression and physiological role of CCN4/Wnt-induced secreted protein 1 mRNA splicing variants in chondrocytes. - sprookjes - yvandenbroek - TriplyDB

Expression and physiological role of CCN4/Wnt-induced secreted protein 1 mRNA splicing variants in chondrocytes.

Expression and physiological role of CCN4/Wnt-induced secreted protein 1 mRNA splicing variants in chondrocytes.

http://www.wikidata.org/entity/Q40155080

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Novel effects of CCN3 that may direct the differentiation of chondrocytes Eyeing the Cyr61/CTGF/NOV (CCN) group of genes in development and diseases: highlights of their structural likenesses and functional dissimilarities Cooperative regulation of chondrocyte differentiation by CCN2 and CCN3 shown by a comprehensive analysis of the CCN family proteins in cartilage The CCN proteins: important signaling mediators in stem cell differentiation and tumorigenesis.Association of the metastatic phenotype with CCN family members among breast and oral cancer cells.CCN3 modulates bone turnover and is a novel regulator of skeletal metastasis Role of WNT1-inducible-signaling pathway protein 1 in etoposide resistance in lung adenocarcinoma A549 cells Posttranscriptional regulation of chicken ccn2 gene expression by nucleophosmin/B23 during chondrocyte differentiation TGF-beta1 and WISP-1/CCN-4 can regulate each other's activity to cooperatively control osteoblast function The role of CCN family genes in haematological malignancies.Dual effect of WISP-1 in diverse pathological processes.Immunohistochemical Analysis of CCN Proteins in Calcified Tissues.CCN3-mutant mice are distinct from CCN3-null mice.A functional analysis of Wnt inducible signalling pathway protein -1 (WISP-1/CCN4).Functional requirement of CCN2 for intramembranous bone formation in embryonic mice.Alternative splicing of CCN mRNAs .... it has been upon us.Roles of heparan sulfate sulfation in dentinogenesis Domain-specific CCN3 antibodies as unique tools for structural and functional studies.CCN3-mediated promotion of sulfated proteoglycan synthesis in rat chondrocytes from developing joint heads.CCN4/WISP-1 positively regulates chondrogenesis by controlling TGF-β3 function.Assessment of circulating Wnt1 inducible signalling pathway protein 1 (WISP-1)/CCN4 as a novel biomarker of obesity.Preparation of Module-Specific Antibodies Against CCN Family Members.Regulation of chondrocytic phenotype by micro RNA 18a: involvement of Ccn2/Ctgf as a major target gene.The role of CCN4/WISP-1 in the cancerous phenotype

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Expression and physiological role of CCN4/Wnt-induced secreted protein 1 mRNA splicing variants in chondrocytes.

http://www.wikidata.org/entity/Q40155080

description

2007 nî lūn-bûn

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Expression and physiological r ...... cing variants in chondrocytes.

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Expression and physiological r ...... cing variants in chondrocytes.

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Expression and physiological r ...... cing variants in chondrocytes.

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J.1742-4658.2007.05709.X

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Expression and physiological r ...... icing variants in chondrocytes

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Harumi Kawaki

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Kazumi Kawata

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Masaharu Takigawa

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Satoshi Kubota

http://www.w3.org/2001/XMLSchema#string

Shinji Tanaka

http://www.w3.org/2001/XMLSchema#string

Takeshi Yanagita

http://www.w3.org/2001/XMLSchema#string

Teruko Takano-Yamamoto

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P2860

WISP genes are members of the connective tissue growth factor family that are up-regulated in wnt-1-transformed cells and aberrantly expressed in human colon tumors

Proposal for a unified CCN nomenclature.

CYR61, a product of a growth factor-inducible immediate early gene, promotes angiogenesis and tumor growth

Inhibition of CCN6 (WISP3) expression promotes neoplastic progression and enhances the effects of insulin-like growth factor-1 on breast epithelial cells.

NOV (nephroblastoma overexpressed) and the CCN family of genes: structural and functional issues.

Overexpression of connective tissue growth factor/hypertrophic chondrocyte-specific gene product 24 decreases bone density in adult mice and induces dwarfism

Identification of a novel integrin alpha 6 beta 1 binding site in the angiogenic inducer CCN1 (CYR61)

A novel variant of WISP1 lacking a Von Willebrand type C module overexpressed in scirrhous gastric carcinoma.

WISP-1 binds to decorin and biglycan.

Overexpression of WISP-1 down-regulated motility and invasion of lung cancer cells through inhibition of Rac activation.

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