Resistance to Pseudomonas aeruginosa chronic lung infection requires cystic fibrosis transmembrane conductance regulator-modulated interleukin-1 (IL-1) release and signaling through the IL-1 receptor.
about
Toll-like receptors in the host defense against Pseudomonas aeruginosa respiratory infection and cystic fibrosisInflammasome-mediated IL-1β production in humans with cystic fibrosisCFTR is a negative regulator of NFkappaB mediated innate immune response.Pseudomonas aeruginosa LPS or flagellin are sufficient to activate TLR-dependent signaling in murine alveolar macrophages and airway epithelial cells.Disruption of interleukin-1β autocrine signaling rescues complex I activity and improves ROS levels in immortalized epithelial cells with impaired cystic fibrosis transmembrane conductance regulator (CFTR) function.Role of Interleukin-17 in defense against pseudomonas aeruginosa infection in lungs.Caveolin-1 modifies the immunity to Pseudomonas aeruginosaPseudomonas aeruginosa triggers CFTR-mediated airway surface liquid secretion in swine trachea.siRNA-mediated gene silencing of MexB from the MexA-MexB-OprM efflux pump in Pseudomonas aeruginosaInterleukin-1 receptor signaling is required to overcome the effects of pertussis toxin and for efficient infection- or vaccination-induced immunity against Bordetella pertussis.The NF-kappaB signaling in cystic fibrosis lung disease: pathophysiology and therapeutic potentialRole of Toll interleukin-1 receptor (IL-1R) 8, a negative regulator of IL-1R/Toll-like receptor signaling, in resistance to acute Pseudomonas aeruginosa lung infection.Autophagy stimulation by rapamycin suppresses lung inflammation and infection by Burkholderia cenocepacia in a model of cystic fibrosis.Immune recognition of Pseudomonas aeruginosa mediated by the IPAF/NLRC4 inflammasome.IL-1 receptor regulates S100A8/A9-dependent keratinocyte resistance to bacterial invasion.Disruption of CFTR-dependent lipid rafts reduces bacterial levels and corneal disease in a murine model of Pseudomonas aeruginosa keratitisIL-1 receptor antagonist ameliorates inflammasome-dependent inflammation in murine and human cystic fibrosisAirway epithelial control of Pseudomonas aeruginosa infection in cystic fibrosis.IL1B polymorphisms modulate cystic fibrosis lung disease.Hyperinflammation in airways of cystic fibrosis patients: what's new?Cystic fibrosis transmembrane conductance regulator and caveolin-1 regulate epithelial cell internalization of Pseudomonas aeruginosa.Inescapable need for neutrophils as mediators of cellular innate immunity to acute Pseudomonas aeruginosa pneumonia.The Pseudomonas aeruginosa Type III secretion system plays a dual role in the regulation of caspase-1 mediated IL-1beta maturation.Bacterial Secretant from Pseudomonas aeruginosa Dampens Inflammasome Activation in a Quorum Sensing-Dependent Manner.The challenges and promises of new therapies for cystic fibrosis.Structure and function of the Type III secretion system of Pseudomonas aeruginosa.Clinical Isolates of Pseudomonas aeruginosa from Chronically Infected Cystic Fibrosis Patients Fail To Activate the Inflammasome during Both Stable Infection and Pulmonary Exacerbation.Cytokine-Regulation of Na+-K+-Cl- Cotransporter 1 and Cystic Fibrosis Transmembrane Conductance Regulator-Potential Role in Pulmonary Inflammation and Edema Formation.N-glycosylation augmentation of the cystic fibrosis epithelium improves Pseudomonas aeruginosa clearance.Cigarette smoke synergizes lipopolysaccharide-induced interleukin-1β and tumor necrosis factor-α secretion from macrophages via substance P-mediated nuclear factor-κB activation.T3SS effector ExoY reduces inflammasome-related responses by suppressing bacterial motility and delaying activation of NF-κB and caspase-1.Interleukin-23-mediated inflammation in Pseudomonas aeruginosa pulmonary infection.Identification of Immune Effectors Essential to the Control of Primary and Secondary Intranasal Infection with Brucella melitensis in Mice.Naturally produced outer membrane vesicles from Pseudomonas aeruginosa elicit a potent innate immune response via combined sensing of both lipopolysaccharide and protein components.Cystic fibrosis swine fail to secrete airway surface liquid in response to inhalation of pathogens.Pseudomonas aeruginosa: can studies in engineered cells tell us why is it such a problem in people with cystic fibrosis? Focus on "Cystic fibrosis transmembrane conductance regulator and caveolin-1 regulate epithelial cell internalization of Pseudom
P2860
Q27010697-7093A154-517A-4FCE-A707-58F20DA38F80Q28483902-9001B9ED-BD6C-4B4D-AB87-A86555AC717FQ33413105-8A9BD136-2E83-49B2-AC72-6F611A219600Q33508834-28F68081-3A1B-47B3-8E4F-73CED9113FA8Q33717303-AFA346DB-D084-49CE-BE1B-4A350F133CCDQ33756193-98829994-970D-4C86-B7E0-898022D0BB02Q33979216-8C694092-2C1F-43DA-AF51-6BD52AB71FFCQ34144306-81C60F4C-6E48-4C7F-81C7-847F768B53D4Q34180060-C7052817-2A87-41FA-B3DD-2490493CB8A7Q34484529-48B05756-1ECE-41CC-A0DE-FDBDDB6C3A89Q35043484-A57734F6-683F-4E21-AC61-8A91A099EDD3Q35665349-C2AEB0D3-5692-4C71-97FF-986A3DEE5C1AQ35985429-04BC7172-DC14-4FD4-8A87-3B96C5101493Q36294419-165E7047-0A97-463B-874E-1AD836F7227CQ36334024-4B80847A-A3A9-4D82-9603-FC9E5A565929Q36517801-8E541D7F-6053-4D0E-B9A7-F0BD24878549Q36690556-242B4141-6E9F-49D4-8782-484CFFB7FEF8Q36968815-DF3618F6-DB1E-4482-AD39-BD84D3748038Q37027691-B74E341A-643A-41A3-BD77-8C2A40D512CBQ37206228-3E4725E7-CB2E-4483-BF82-81AF8377E94EQ37298019-2C62FD97-484A-4AA2-9BF1-A7F310140D94Q37451128-E8768FC0-2F72-4ECA-BEA3-DC4DDD8E590EQ37568779-F46B1083-24F1-412D-9D0B-FEBDAE0CF153Q37721547-0B6FD213-D3D5-4440-AB15-CFFF685E39D6Q38023386-BDDF4EF1-2D21-46D2-9EA4-0C555E61AD84Q38072996-3EAB2F8A-FECC-4A40-A165-9593E1A9ADD0Q38792565-F57E8DB4-F625-43D8-A0A3-39977AEBA080Q39260011-492B2B25-939F-4325-8523-05C23657B366Q39669861-9BD8D322-20A7-4C92-AF88-D1F4F337EB20Q39738730-DCF2AF6E-114C-4000-9E02-0FA7C0345A22Q40090334-1439A1A2-9D53-4C1C-8BA5-909CD0817E76Q40313360-30B11BC3-54EE-4758-9F88-36DDA9E78919Q40731935-30DDFFC9-5522-4B44-9003-A5C3E12A7DCEQ42181392-2DDFF854-0721-4550-8826-7A5E7A5AD479Q42277379-409E7DD6-E29F-41BD-B714-A0F9251DE16EQ53387514-8ABE6658-2BB8-4A01-BDC9-80D711E789E4
P2860
Resistance to Pseudomonas aeruginosa chronic lung infection requires cystic fibrosis transmembrane conductance regulator-modulated interleukin-1 (IL-1) release and signaling through the IL-1 receptor.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Resistance to Pseudomonas aeru ...... ing through the IL-1 receptor.
@en
type
label
Resistance to Pseudomonas aeru ...... ing through the IL-1 receptor.
@en
prefLabel
Resistance to Pseudomonas aeru ...... ing through the IL-1 receptor.
@en
P2093
P2860
P921
P356
P1476
Resistance to Pseudomonas aeru ...... ing through the IL-1 receptor.
@en
P2093
Christopher Ray
David E Golan
Fadie T Coleman
Martin M Lee
Nina Reiniger
P2860
P304
P356
10.1128/IAI.01980-06
P407
P50
P577
2007-02-05T00:00:00Z