Acid increases MAPK-mediated proliferation in Barrett's esophageal adenocarcinoma cells via intracellular acidification through a Cl-/HCO3- exchanger.
about
Activation of Akt is increased in the dysplasia-carcinoma sequence in Barrett's oesophagus and contributes to increased proliferation and inhibition of apoptosis: a histopathological and functional studyGene expression changes associated with Barrett's esophagus and Barrett's-associated adenocarcinoma cell lines after acid or bile salt exposure.Expression and role of anion exchanger 1 in esophageal squamous cell carcinomaRole of the Na ⁺/K ⁺/2Cl⁻ cotransporter NKCC1 in cell cycle progression in human esophageal squamous cell carcinoma.Characterization of squamous esophageal cells resistant to bile acids at acidic pH: implication for Barrett's esophagus pathogenesisBile acids in combination with low pH induce oxidative stress and oxidative DNA damage: relevance to the pathogenesis of Barrett's oesophagus.Role of chemoprophylaxis with either NSAIDs or statins in patients with Barrett's esophagus.Extracellular acidity, a "reappreciated" trait of tumor environment driving malignancy: perspectives in diagnosis and therapy.Intracellular chloride regulates the G(1)/S cell cycle progression in gastric cancer cells.The acidic microenvironment as a possible niche of dormant tumor cells.Physiological and molecular analysis of acid loading mechanisms in squamous and columnar-lined esophagus.Acid has antiproliferative effects in nonneoplastic Barrett's epithelial cells.COX-2 induction by unconjugated bile acids involves reactive oxygen species-mediated signalling pathways in Barrett's oesophagus and oesophageal adenocarcinoma.Intrinsic resistance triggered under acid loading within normal esophageal epithelial cells: NHE1- and ROS-mediated survival.Carbonic anhydrase II regulates differentiation of ameloblasts via intracellular pH-dependent JNK signaling pathway.Bicarbonate transport inhibitor SITS modulates pH homeostasis triggering apoptosis of Dalton's lymphoma: implication of novel molecular mechanisms.Activity of mitogen-activated protein kinases in the esophageal epithelium of patients with Barrett's esophagus.The mechanism of MAP kinase activation under acidic condition in feline esophageal smooth muscle cells.Cell culture models for studying the development of Barrett's esophagus: a systematic review.
P2860
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P2860
Acid increases MAPK-mediated proliferation in Barrett's esophageal adenocarcinoma cells via intracellular acidification through a Cl-/HCO3- exchanger.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
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2005年學術文章
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name
Acid increases MAPK-mediated p ...... through a Cl-/HCO3- exchanger.
@en
type
label
Acid increases MAPK-mediated p ...... through a Cl-/HCO3- exchanger.
@en
prefLabel
Acid increases MAPK-mediated p ...... through a Cl-/HCO3- exchanger.
@en
P2093
P2860
P356
P1476
Acid increases MAPK-mediated p ...... through a Cl-/HCO3- exchanger.
@en
P2093
Christie Lopez-Guzman
Emily Herndon
George A Sarosi
Kshama Jaiswal
Rhonda F Souza
Stuart J Spechler
P2860
P304
P356
10.1152/AJPGI.00215.2005
P577
2005-08-04T00:00:00Z