The basal proton conductance of mitochondria depends on adenine nucleotide translocase content.
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Uncoupling protein-4 (UCP4) increases ATP supply by interacting with mitochondrial Complex II in neuroblastoma cellsMolecular aspects of thyroid hormone actionsPathogenic VCP mutations induce mitochondrial uncoupling and reduced ATP levelsMitochondria in the diabetic heartThe Measurement of Reversible Redox Dependent Post-translational Modifications and Their Regulation of Mitochondrial and Skeletal Muscle FunctionThyroid Hormone Mediated Modulation of Energy ExpenditureCell Biology Symposium: feed efficiency: mitochondrial function to global gene expressionImplications of mitochondrial uncoupling in skeletal muscle in the development and treatment of obesityThe Secreted Enzyme PM20D1 Regulates Lipidated Amino Acid Uncouplers of MitochondriaMitochondrial proton and electron leaksPhysiological and pathological roles of mitochondrial SLC25 carriersMitochondrial Metabolism in Aging HeartElevated mitochondrial oxidative stress impairs metabolic adaptations to exercise in skeletal muscleEnergization-dependent endogenous activation of proton conductance in skeletal muscle mitochondriaPhenotypic rescue of a Drosophila model of mitochondrial ANT1 disease.Hyperglycemia alters the schwann cell mitochondrial proteome and decreases coupled respiration in the absence of superoxide production.Adenine nucleotide translocase 1 deficiency results in dilated cardiomyopathy with defects in myocardial mechanics, histopathological alterations, and activation of apoptosis.The mitochondrial complex II and ATP-sensitive potassium channel interaction: quantitation of the channel in heart mitochondria.Electron transport chain-dependent and -independent mechanisms of mitochondrial H2O2 emission during long-chain fatty acid oxidation.Mutations in valosin-containing protein (VCP) decrease ADP/ATP translocation across the mitochondrial membrane and impair energy metabolism in human neurons.Adenine nucleotide translocase is acetylated in vivo in human muscle: Modeling predicts a decreased ADP affinity and altered control of oxidative phosphorylation.Role of the transmembrane potential in the membrane proton leak.The regulation and turnover of mitochondrial uncoupling proteins.Control mechanisms in mitochondrial oxidative phosphorylation.Uncoupling protein-3 lowers reactive oxygen species production in isolated mitochondria.The on-off switches of the mitochondrial uncoupling proteins.The biochemical properties of the mitochondrial thiamine pyrophosphate carrier from Drosophila melanogaster.Nitroalkenes confer acute cardioprotection via adenine nucleotide translocase 1.Computational modelling of the piglet brain to simulate near-infrared spectroscopy and magnetic resonance spectroscopy data collected during oxygen deprivation.A novel high-throughput assay for islet respiration reveals uncoupling of rodent and human islets.Unraveling biochemical pathways affected by mitochondrial dysfunctions using metabolomic approaches.Acute knockdown of uncoupling protein-2 increases uncoupling via the adenine nucleotide transporter and decreases oxidative stress in diabetic kidneysMitochondrial reactive oxygen species modulate mosquito susceptibility to Plasmodium infection.Calorie restriction in mice overexpressing UCP3: evidence that prior mitochondrial uncoupling alters response.Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.Different effects of guanine nucleotides (GDP and GTP) on protein-mediated mitochondrial proton leak.Caenorhabditis elegans UCP4 protein controls complex II-mediated oxidative phosphorylation through succinate transport.Adenine nucleotide translocator 1 deficiency increases resistance of mouse brain and neurons to excitotoxic insults.Cytosolic, but not mitochondrial, oxidative stress is a likely contributor to cardiac hypertrophy resulting from cardiac specific GLUT4 deletion in miceMisfolding of mutant adenine nucleotide translocase in yeast supports a novel mechanism of Ant1-induced muscle diseases.
P2860
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P2860
The basal proton conductance of mitochondria depends on adenine nucleotide translocase content.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
The basal proton conductance o ...... ucleotide translocase content.
@en
type
label
The basal proton conductance o ...... ucleotide translocase content.
@en
prefLabel
The basal proton conductance o ...... ucleotide translocase content.
@en
P2093
P2860
P356
P1433
P1476
The basal proton conductance o ...... ucleotide translocase content.
@en
P2093
Douglas C Wallace
Emma J Cornwall
Jason Kokoszka
Julian L Pakay
Paul S Brookes
P2860
P304
P356
10.1042/BJ20050890
P407
P577
2005-12-01T00:00:00Z