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Unraveling the complex metabolic nature of astrocytesDeterminants of brain cell metabolic phenotypes and energy substrate utilization unraveled with a modeling approachSimilar perisynaptic glial localization for the Na+,K+-ATPase alpha 2 subunit and the glutamate transporters GLAST and GLT-1 in the rat somatosensory cortexResistance to diet-induced obesity and associated metabolic perturbations in haploinsufficient monocarboxylate transporter 1 miceCellular mechanisms of brain energy metabolism and their relevance to functional brain imaging.Feeding active neurons: (re)emergence of a nursing role for astrocytes.Metabolic compartmentalization in the human cortex and hippocampus: evidence for a cell- and region-specific localization of lactate dehydrogenase 5 and pyruvate dehydrogenase.Glial glutamate transporters mediate a functional metabolic crosstalk between neurons and astrocytes in the mouse developing cortex.GABA uptake into astrocytes is not associated with significant metabolic cost: implications for brain imaging of inhibitory transmission.Oligodendroglia metabolically support axons and contribute to neurodegeneration.Glutamate reduces glucose utilization while concomitantly enhancing AQP9 and MCT2 expression in cultured rat hippocampal neuronsThe astrocyte-mediated coupling between synaptic activity and energy metabolism operates through volume transmission.Brain lactate kinetics: Modeling evidence for neuronal lactate uptake upon activationBrain-derived neurotrophic factor enhances the expression of the monocarboxylate transporter 2 through translational activation in mouse cultured cortical neurons.Rise in plasma lactate concentrations with psychosocial stress: a possible sign of cerebral energy demand.Activity-dependent regulation of energy metabolism by astrocytes: an update.Alzheimer's disease: the amyloid hypothesis and the Inverse Warburg effect.Role of neuron-glia interaction in the regulation of brain glucose utilization.Lactate as a pivotal element in neuron-glia metabolic cooperation.Neuroenergetic Response to Prolonged Cerebral Glucose Depletion after Severe Brain Injury and the Role of Lactate.Caveolin expression changes in the neurovascular unit after juvenile traumatic brain injury: signs of blood-brain barrier healing?A coherent neurobiological framework for functional neuroimaging provided by a model integrating compartmentalized energy metabolism.Neuroenergetics: calling upon astrocytes to satisfy hungry neurons.Expression of monocarboxylate transporter mRNAs in mouse brain: support for a distinct role of lactate as an energy substrate for the neonatal vs. adult brainSweet sixteen for ANLS.Monocarboxylate transporters in the central nervous system: distribution, regulation and function.How astrocytes feed hungry neurons.A probable dual mode of action for both L- and D-lactate neuroprotection in cerebral ischemia.A neuronal MCT2 knockdown in the rat somatosensory cortex reduces both the NMR lactate signal and the BOLD response during whisker stimulationImprovement of Neuroenergetics by Hypertonic Lactate Therapy in Patients with Traumatic Brain Injury Is Dependent on Baseline Cerebral Lactate/Pyruvate Ratio.Temporal changes in mRNA expression of the brain nutrient transporters in the lithium-pilocarpine model of epilepsy in the immature and adult rat.Monocarboxylate transporters in the brain and in cancer.E4F1-mediated control of pyruvate dehydrogenase activity is essential for skin homeostasis.Brain energetics (thought needs food).Hypothalamic sensing of ketone bodies after prolonged cerebral exposure leads to metabolic control dysregulation.Cerebral Ketone Body Oxidation Is Facilitated by a High Fat Diet Enriched with Advanced Glycation End Products in Normal and Diabetic Rats.Food for thought: the importance of glucose and other energy substrates for sustaining brain function under varying levels of activity.Enhanced expression of three monocarboxylate transporter isoforms in the brain of obese mice.Local injection of antisense oligonucleotides targeted to the glial glutamate transporter GLAST decreases the metabolic response to somatosensory activation.Monocarboxylate transporters: new players in body weight regulation.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Luc Pellerin
@ast
Luc Pellerin
@en
Luc Pellerin
@es
Luc Pellerin
@nl
Luc Pellerin
@sl
type
label
Luc Pellerin
@ast
Luc Pellerin
@en
Luc Pellerin
@es
Luc Pellerin
@nl
Luc Pellerin
@sl
prefLabel
Luc Pellerin
@ast
Luc Pellerin
@en
Luc Pellerin
@es
Luc Pellerin
@nl
Luc Pellerin
@sl
P1053
A-8912-2017
P106
P21
P31
P3829
P3835
luc-pellerin2
P496
0000-0002-1016-1970