Application of IL-36 receptor antagonist weakens CCL20 expression and impairs recovery in the late phase of murine acetaminophen-induced liver injury.
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STAT3, a Key Parameter of Cytokine-Driven Tissue Protection during Sterile Inflammation - the Case of Experimental Acetaminophen (Paracetamol)-Induced Liver DamageType I Interferon Supports Inducible Nitric Oxide Synthase in Murine Hepatoma Cells and Hepatocytes and during Experimental Acetaminophen-Induced Liver Damage.Distinct expression of interleukin (IL)-36α, β and γ, their antagonist IL-36Ra and IL-38 in psoriasis, rheumatoid arthritis and Crohn's disease.IL-36γ has proinflammatory effects on human endothelial cells.Cell death and inflammation: the case for IL-1 family cytokines as the canonical DAMPs of the immune system.Autocrine and Paracrine Regulation of Keratinocyte Proliferation through a Novel Nrf2-IL-36γ Pathway.Selective targeting of tumor associated macrophages in different tumor models.A Prominent Role of Interleukin-18 in Acetaminophen-Induced Liver Injury Advocates Its Blockage for Therapy of Hepatic Necroinflammation.
P2860
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P2860
Application of IL-36 receptor antagonist weakens CCL20 expression and impairs recovery in the late phase of murine acetaminophen-induced liver injury.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Application of IL-36 receptor ...... minophen-induced liver injury.
@en
type
label
Application of IL-36 receptor ...... minophen-induced liver injury.
@en
prefLabel
Application of IL-36 receptor ...... minophen-induced liver injury.
@en
P2093
P2860
P356
P1433
P1476
Application of IL-36 receptor ...... minophen-induced liver injury.
@en
P2093
Bernhard Zwissler
Heiko Mühl
Josef Pfeilschifter
Lorena Härdle
Malte Bachmann
Patrick Scheiermann
Thomas Pleli
P2860
P2888
P356
10.1038/SREP08521
P407
P577
2015-02-17T00:00:00Z