about
Metaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injuryGlial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learningBrain-derived neurotrophic factor promotes adaptive plasticity within the spinal cord and mediates the beneficial effects of controllable stimulationIntrathecal administration of neurokinin 1 and neurokinin 2 receptor antagonists undermines the savings effect in spinal rats seen in an instrumental learning paradigmHemodynamic responses to speech and music in preverbal infants.Timing in the absence of supraspinal input II: regularly spaced stimulation induces a lasting alteration in spinal function that depends on the NMDA receptor, BDNF release, and protein synthesisKeratinocytes can modulate and directly initiate nociceptive responses.Learning about time within the spinal cord: evidence that spinal neurons can abstract and store an index of regularity.Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury.Managing Chronic Pain in Special Populations with Emphasis on Pediatric, Geriatric, and Drug Abuser PopulationsPain and learning in a spinal system: contradictory outcomes from common origins.Semi-intact ex vivo approach to investigate spinal somatosensory circuits.Neurobiological Consequences of Early Painful Experience: Basic Science Findings and Implications for Evidence-Based Practice.BDNF and learning: Evidence that instrumental training promotes learning within the spinal cord by up-regulating BDNF expression.Deletion of the murine ATP/UTP receptor P2Y2 alters mechanical and thermal response properties in polymodal cutaneous afferents.Timing in the absence of supraspinal input I: variable, but not fixed, spaced stimulation of the sciatic nerve undermines spinally-mediated instrumental learning.Spinal glia modulate both adaptive and pathological processes.Temporal regularity determines the impact of electrical stimulation on tactile reactivity and response to capsaicin in spinally transected rats.Cutaneous neurturin overexpression alters mechanical, thermal, and cold responsiveness in physiologically identified primary afferents.Timing in the absence of supraspinal input III: regularly spaced cutaneous stimulation prevents and reverses the spinal learning deficit produced by peripheral inflammation.Local anesthetic treatment significantly attenuates acute pain responding but does not prevent the neonatal injury-induced reduction in adult spinal behavioral plasticity.Variations in COMT and NTRK2 Influence Symptom Burden in Women Undergoing Breast Cancer Treatment.Factors Leading to Persistent Postsurgical Pain in Adolescents Undergoing Spinal Fusion: An Integrative Literature Review.Lipopolysaccharide induces a spinal learning deficit that is blocked by IL-1 receptor antagonism.The neonatal injury-induced spinal learning deficit in adult rats: central mechanisms.Intrathecal infusions of anisomycin impact the learning deficit but not the learning effect observed in spinal rats that have received instrumental training.Neonatal hind-paw injury disrupts acquisition of an instrumental response in adult spinal rats.Fixed spaced stimulation restores adaptive plasticity within the spinal cord: Identifying the eliciting conditions.Neurokinin receptors modulate the impact of uncontrollable stimulation on adaptive spinal plasticity.Administration of a Ca-super(2+)/calmodulin-dependent protein kinase II (CaMKII) inhibitor prevents the learning deficit observed in spinal rats after noncontingent shock administration.Injectable nanocomposite analgesic delivery system for musculoskeletal pain management
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P50
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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Kyle M. Baumbauer
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P106
P1153
14319126200
P2798
P31
P496
0000-0003-0437-9209