Double-strand breaks induce homologous recombinational repair of interstrand cross-links via cooperation of MSH2, ERCC1-XPF, REV3, and the Fanconi anemia pathway.
about
FANCG promotes formation of a newly identified protein complex containing BRCA2, FANCD2 and XRCC3Cellular and molecular consequences of defective Fanconi anemia proteins in replication-coupled DNA repair: mechanistic insightsGenetic Variants That Predispose to DNA Double-Strand Breaks in Lymphocytes From a Subset of Patients With Familial Colorectal CarcinomasStructural, molecular and cellular functions of MSH2 and MSH6 during DNA mismatch repair, damage signaling and other noncanonical activitiesOligo/polynucleotide-based gene modification: strategies and therapeutic potentialExpanded roles of the Fanconi anemia pathway in preserving genomic stabilityThe role of the Fanconi anemia network in the response to DNA replication stress.Mismatch repair and nucleotide excision repair proteins cooperate in the recognition of DNA interstrand crosslinks.Repair of laser-localized DNA interstrand cross-links in G1 phase mammalian cellsDifferential roles for DNA polymerases eta, zeta, and REV1 in lesion bypass of intrastrand versus interstrand DNA cross-links.DNA interstrand crosslink repair in mammalian cells: step by step.Targeting and processing of site-specific DNA interstrand crosslinks.The Fanconi anemia protein, FANCG, binds to the ERCC1-XPF endonuclease via its tetratricopeptide repeats and the central domain of ERCC1.Initiation of DNA interstrand cross-link repair in mammalian cells.Regulation of DNA cross-link repair by the Fanconi anemia/BRCA pathwayLoss of DNA polymerase zeta enhances spontaneous tumorigenesis.MSH3 mediates sensitization of colorectal cancer cells to cisplatin, oxaliplatin, and a poly(ADP-ribose) polymerase inhibitor.Homology-directed Fanconi anemia pathway cross-link repair is dependent on DNA replication.Contributions of DNA interstrand cross-links to aging of cells and organisms.Fanconi anemia proteins, DNA interstrand crosslink repair pathways, and cancer therapy.DNA polymerases and cancer.Effect of cross-link structure on DNA interstrand cross-link repair synthesisDNA polymerase zeta is essential for hexavalent chromium-induced mutagenesis.Targeted gene conversion induced by triplex-directed psoralen interstrand crosslinks in mammalian cells.Repair of DNA interstrand cross-links during S phase of the mammalian cell cycle.Translesion DNA synthesis in the context of cancer research.The potential of exploiting DNA-repair defects for optimizing lung cancer treatment.REV1 and DNA polymerase zeta in DNA interstrand crosslink repair.The roles of DNA polymerase ζ and the Y family DNA polymerases in promoting or preventing genome instability.Low-fidelity compensatory backup alternative DNA repair pathways may unify current carcinogenesis theories.Functional and physical interaction between the mismatch repair and FA-BRCA pathways.c-Myc-miR-29c-REV3L signalling pathway drives the acquisition of temozolomide resistance in glioblastoma.Crosstalk between BRCA-Fanconi anemia and mismatch repair pathways prevents MSH2-dependent aberrant DNA damage responsesThe ERCC1/XPF endonuclease is required for completion of homologous recombination at DNA replication forks stalled by inter-strand cross-links.Impaired removal of DNA interstrand cross-link in Nijmegen breakage syndrome and Fanconi anemia, but not in BRCA-defective group.The Homologous Recombination Machinery Orchestrates Post-replication DNA Repair During Self-renewal of Mouse Embryonic Stem Cells.Mechanism of RAD51-dependent DNA interstrand cross-link repair.Snm1B/Apollo mediates replication fork collapse and S Phase checkpoint activation in response to DNA interstrand cross-links.Roles of human POLD1 and POLD3 in genome stability.3-Methyladenine DNA glycosylase is important for cellular resistance to psoralen interstrand cross-links.
P2860
Q24307531-77C4394A-6907-44F4-9835-0D4662196096Q28252640-9382A873-587B-45D0-9E7F-3AF4E8C39824Q28267038-E8688772-F68F-4AB0-83A7-44A3745CA84BQ28285134-7C2F60AC-6192-4712-9F8E-17520EF0F6D6Q28307657-9367F5E7-44BF-49B8-9713-26E24604D975Q28749668-5C316FD6-D326-4B23-83A0-2562E38D8528Q33347783-84A6929B-C61D-4D56-9E4B-FD1A56CFC523Q33452435-9FD03AB9-E746-43A5-A41B-574A6A3D3748Q33493863-FFCCD9C3-52D4-416F-AF4B-68FF876555CEQ33648841-63340475-E732-4F90-BDBE-DEA09454FD3BQ33669915-6B7719C3-20FA-4F83-BF78-54CDF2BB07FBQ33952673-8C274571-3C47-4387-907D-E4D50C663F59Q34000660-2C611816-28B3-413B-90CC-75DF20100667Q34026606-517AB507-04BF-42D6-9292-2521DB566F89Q34285356-E93BF442-7590-4E42-9A6C-4458DE7A7966Q34432676-D40E59F8-2D0B-4A52-8B29-7CDD422B053BQ34752116-678B667D-4516-4421-908A-BFBF44C12626Q35741074-C34B88BD-1DC6-480C-B85B-8127873BF6E0Q37033646-B688FE5C-35A0-4161-8C34-CC7873E02D8BQ37069628-64C8AC24-AEE7-4B83-ADC4-DEC583F1FFABQ37084895-68A03A9F-EF5B-45FB-92ED-AED84B917046Q37352226-9501E2A1-22D5-46D1-979E-D2D0D94EDE33Q37365913-81F62088-FBF9-4F0F-9CB3-D156B2C1F082Q37406060-EAEBE002-B82F-47AF-B470-D7C187E38105Q37775586-0A050907-BD76-4493-B759-581E65893EF7Q37952001-244651F1-7441-4280-AACF-6D063977F442Q37983690-C4FE3B6F-0D59-42A3-B035-405AF30EC6E4Q38052100-8CA24C6C-D622-4C01-9E24-33D80ECBA60DQ38063711-B8F0990D-0D0D-4F8C-82B8-80BEAD100D76Q38221690-33C43CDA-CB4B-4C6B-AF44-1224597268D2Q38755812-8DB31298-C89A-4A76-8432-2E600C9AA95FQ38829331-563162A9-23A1-43CE-B884-376C555F0DA7Q38981181-9BDAF148-E69F-4AFF-8E77-1358B2B1DC2AQ39807128-6EE72C99-9C0F-4FEE-BF30-AC027C1535E5Q39943294-14A1AAF1-83BD-4775-9AF5-BA9DF7AF2F00Q41047419-8234CFC2-1BF6-4FFB-8CFA-022A1FFFE74FQ41070024-61A6412E-47A1-4DEB-9C75-66039E96869EQ42044832-0FBBFA9F-02FD-4FCD-8E87-C3FDF74B11B9Q42104410-9A29EAEC-9E96-40F1-9486-06687E5756F9Q42709830-AAB4B317-D2BF-4C6B-B885-FA1A758AE410
P2860
Double-strand breaks induce homologous recombinational repair of interstrand cross-links via cooperation of MSH2, ERCC1-XPF, REV3, and the Fanconi anemia pathway.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Double-strand breaks induce ho ...... nd the Fanconi anemia pathway.
@en
type
label
Double-strand breaks induce ho ...... nd the Fanconi anemia pathway.
@en
prefLabel
Double-strand breaks induce ho ...... nd the Fanconi anemia pathway.
@en
P2093
P2860
P1433
P1476
Double-strand breaks induce ho ...... nd the Fanconi anemia pathway.
@en
P2093
Nianxiang Zhang
Randy Legerski
Xiuping Liu
P2860
P304
P356
10.1016/J.DNAREP.2007.06.002
P577
2007-07-31T00:00:00Z