Indirect Suppression Involving Behavioral Mutants with Altered Nerve Excitability in DROSOPHILA MELANOGASTER.
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Structural and Genetic Studies Demonstrate Neurologic Dysfunction in Triosephosphate Isomerase Deficiency Is Associated with Impaired Synaptic Vesicle DynamicsThe Microtubule Regulatory Protein Stathmin Is Required to Maintain the Integrity of Axonal Microtubules in DrosophilaModes of metabolic compensation during mitochondrial disease using the Drosophila model of ATP6 dysfunctionOxidative stress is not a major contributor to somatic mitochondrial DNA mutationsSDHAF4 promotes mitochondrial succinate dehydrogenase activity and prevents neurodegeneration.Circadian Clock Dysfunction and Psychiatric Disease: Could Fruit Flies have a Say?Drosophila sodium channel mutations: Contributions to seizure-susceptibilityCalcium Imaging of Neuronal Activity in Drosophila Can Identify Anticonvulsive CompoundsMutations of the Calcium Channel Gene cacophony Suppress Seizures in DrosophilaMutation of orthologous prickle genes causes a similar epilepsy syndrome in flies and humans.Molecular characterization and expression of the (Na+ + K+)-ATPase alpha-subunit in Drosophila melanogaster.Mutation of Drosophila focal adhesion kinase induces bang-sensitive behavior and disrupts glial function, axonal conduction and synaptic transmission.Courtship and other behaviors affected by a heat-sensitive, molecularly novel mutation in the cacophony calcium-channel gene of DrosophilaTowards a molecular understanding of Drosophila hearingIncreased persistent Na+ current contributes to seizure in the slamdance bang-sensitive Drosophila mutant.A Drosophila model of GSS syndrome suggests defects in active zones are responsible for pathogenesis of GSS syndrome.A Drosophila model of mitochondrial disease caused by a complex I mutation that uncouples proton pumping from electron transfer.Structural mass spectrometry analysis of lipid changes in a Drosophila epilepsy model brain.100 years of Drosophila research and its impact on vertebrate neuroscience: a history lesson for the futureSeizure sensitivity is ameliorated by targeted expression of K+-Cl- cotransporter function in the mushroom body of the Drosophila brain.Multiple functions of Drosophila heat shock transcription factor in vivoThe dissonance mutant of courtship song in Drosophila melanogaster: isolation, behavior and cytogenetics.The Passover locus in Drosophila melanogaster: complex complementation and different effects on the giant fiber neural pathway.Mutation of the axonal transport motor kinesin enhances paralytic and suppresses Shaker in Drosophila.Mutations in the Drosophila pushover gene confer increased neuronal excitability and spontaneous synaptic vesicle fusion.Cloning of the Drosophila melanogaster meiotic recombination gene mei-218: a genetic and molecular analysis of interval 15ECalmodulin point mutations affect Drosophila development and behavior.Drosophila as a model for epilepsy: bss is a gain-of-function mutation in the para sodium channel gene that leads to seizures.Hsp70- and Hsp90-mediated proteasomal degradation underlies TPI sugarkill pathogenesis in Drosophila.Mutations in prickle orthologs cause seizures in flies, mice, and humans.Seizure suppression by gain-of-function escargot mutations.Drosophila couch potato mutants exhibit complex neurological abnormalities including epilepsy phenotypes.Neuropathology in Drosophila membrane excitability mutantsstress sensitive B encodes an adenine nucleotide translocase in Drosophila melanogaster.Triosephosphate isomerase I170V alters catalytic site, enhances stability and induces pathology in a Drosophila model of TPI deficiency.Human disease models in Drosophila melanogaster and the role of the fly in therapeutic drug discovery.Assaying locomotor, learning, and memory deficits in Drosophila models of neurodegeneration.A new model to study sleep deprivation-induced seizure.Drug-resistant Drosophila indicate glutamate-gated chloride channels are targets for the antiparasitics nodulisporic acid and ivermectin.Mutations in cytochrome c oxidase subunit VIa cause neurodegeneration and motor dysfunction in Drosophila
P2860
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P2860
Indirect Suppression Involving Behavioral Mutants with Altered Nerve Excitability in DROSOPHILA MELANOGASTER.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年論文
@yue
1982年論文
@zh-hant
1982年論文
@zh-hk
1982年論文
@zh-mo
1982年論文
@zh-tw
1982年论文
@wuu
1982年论文
@zh
1982年论文
@zh-cn
name
Indirect Suppression Involving ...... ty in DROSOPHILA MELANOGASTER.
@en
type
label
Indirect Suppression Involving ...... ty in DROSOPHILA MELANOGASTER.
@en
prefLabel
Indirect Suppression Involving ...... ty in DROSOPHILA MELANOGASTER.
@en
P2860
P1433
P1476
Indirect Suppression Involving ...... ity in DROSOPHILA MELANOGASTER
@en
P2093
P2860
P304
P407
P577
1982-04-01T00:00:00Z