CDK5 serves as a major control point in neurotransmitter release.
about
LRRK2 phosphorylates Snapin and inhibits interaction of Snapin with SNAP-25Synaptic vesicle pools and dynamicsMolecular Machines Determining the Fate of Endocytosed Synaptic Vesicles in Nerve TerminalsThe emerging roles and therapeutic potential of cyclin-dependent kinase 11 (CDK11) in human cancerMolecular Machines Regulating the Release Probability of Synaptic Vesicles at the Active ZoneMolecular mechanisms driving homeostatic plasticity of neurotransmitter releaseSynaptic vesicle recycling: steps and principlesThe role of ventral striatal cAMP signaling in stress-induced behaviors.Synaptic Vesicle Recycling Is Unaffected in the Ts65Dn Mouse Model of Down SyndromeNeuroprotective Mechanisms Mediated by CDK5 InhibitionActivity-dependent p25 generation regulates synaptic plasticity and Aβ-induced cognitive impairmentActivity-dependent adaptations in inhibitory axonsAnalysis of protein phosphorylation in nerve terminal reveals extensive changes in active zone proteins upon exocytosisCdk5 is a New Rapid Synaptic Homeostasis Regulator Capable of Initiating the Early Alzheimer-Like Pathology.Loss of cyclin-dependent kinase 5 from parvalbumin interneurons leads to hyperinhibition, decreased anxiety, and memory impairmentForebrain-specific deletion of Cdk5 in pyramidal neurons results in mania-like behavior and cognitive impairment.Cdk5 is required for memory function and hippocampal plasticity via the cAMP signaling pathway.Calbindin controls release probability in ventral tegmental area dopamine neurons.Late recruitment of synapsin to nascent synapses is regulated by Cdk5.Synaptic roles of cyclin-dependent kinase 5 & its implications in epilepsyAxonal cap-dependent translation regulates presynaptic p35.Inhibition of exocytosis or endocytosis blocks activity-dependent redistribution of synapsin.Working memory impairment in calcineurin knock-out mice is associated with alterations in synaptic vesicle cycling and disruption of high-frequency synaptic and network activity in prefrontal cortexAt the Fulcrum in Health and Disease: Cdk5 and the Balancing Acts of Neuronal Structure and Physiology.Increased synaptophysin is involved in inflammation-induced heat hyperalgesia mediated by cyclin-dependent kinase 5 in rats.The role of Cdk5 in cognition and neuropsychiatric and neurological pathology.Studying synaptic efficiency by post-hoc immunolabelling.Cannabinoid type 1 receptors transiently silence glutamatergic nerve terminals of cultured cerebellar granule cells.Reduction of phosphorylated synapsin I (ser-553) leads to spatial memory impairment by attenuating GABA release after microwave exposure in Wistar rats.Transsynaptic control of presynaptic Ca²⁺ influx achieves homeostatic potentiation of neurotransmitter release.Targeting cell cycle regulators in hematologic malignancies.A small pool of vesicles maintains synaptic activity in vivo.Cyclin E constrains Cdk5 activity to regulate synaptic plasticity and memory formationConcurrent imaging of synaptic vesicle recycling and calcium dynamicsSynaptic deficits are rescued in the p25/Cdk5 model of neurodegeneration by the reduction of β-secretase (BACE1)A Cdk5-dependent switch regulates Lis1/Ndel1/dynein-driven organelle transport in adult axons.The active-zone protein Munc13 controls the use-dependence of presynaptic voltage-gated calcium channelsRegulation of synaptic activity by snapin-mediated endolysosomal transport and sorting.Molecular pathways associated with the nutritional programming of plant-based diet acceptance in rainbow trout following an early feeding exposure.Snapin is critical for presynaptic homeostatic plasticity
P2860
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P2860
CDK5 serves as a major control point in neurotransmitter release.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
CDK5 serves as a major control point in neurotransmitter release.
@en
type
label
CDK5 serves as a major control point in neurotransmitter release.
@en
prefLabel
CDK5 serves as a major control point in neurotransmitter release.
@en
P2860
P1433
P1476
CDK5 serves as a major control point in neurotransmitter release
@en
P2093
Sung Hyun Kim
Timothy A Ryan
P2860
P304
P356
10.1016/J.NEURON.2010.08.003
P407
P577
2010-09-01T00:00:00Z