The folding of the hepatitis C virus internal ribosome entry site depends on the 3'-end of the viral genome.
about
The yin and yang of hepatitis C: synthesis and decay of hepatitis C virus RNAAn Efficient Microarray-Based Genotyping Platform for the Identification of Drug-Resistance Mutations in Majority and Minority Subpopulations of HIV-1 QuasispeciesA magnesium-induced RNA conformational switch at the internal ribosome entry site of hepatitis C virus genome visualized by atomic force microscopyRNA Interference-Guided Targeting of Hepatitis C Virus Replication with Antisense Locked Nucleic Acid-Based Oligonucleotides Containing 8-oxo-dG Modifications.Netrin-1 Protects Hepatocytes Against Cell Death Through Sustained Translation During the Unfolded Protein ResponseThe non-primate hepacivirus 5' untranslated region possesses internal ribosomal entry site activity.Unmasking the information encoded as structural motifs of viral RNA genomes: a potential antiviral target.Functional long-range RNA-RNA interactions in positive-strand RNA viruses.RNA Aptamers as Molecular Tools to Study the Functionality of the Hepatitis C Virus CRE Region.LOOP IIId of the HCV IRES is essential for the structural rearrangement of the 40S-HCV IRES complex.NMR study of RNA structures in the 3´-end of the Hepatitis C Virus genome.The cis-acting replication element of the Hepatitis C virus genome recruits host factors that influence viral replication and translationThe chaperone-like activity of the hepatitis C virus IRES and CRE elements regulates genome dimerization.Cellular Proteins Act as Bridge Between 5' and 3' Ends of the Coxsackievirus B3 Mediating Genome Circularization During RNA Translation.End-to-end crosstalk within the hepatitis C virus genome mediates the conformational switch of the 3'X-tail region.The 5BSL3.2 Functional RNA Domain Connects Distant Regions in the Hepatitis C Virus Genome.Theory Meets Experiment: Metal Ion Effects in HCV Genomic RNA Kissing Complex Formation.Roles of the 5' untranslated region of non-primate hepacivirus in translation initiation and viral replication.Distinct roles for the IIId2 sub-domain in pestivirus and picornavirus internal ribosome entry sites.
P2860
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P2860
The folding of the hepatitis C virus internal ribosome entry site depends on the 3'-end of the viral genome.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@en
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@nl
type
label
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@en
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@nl
prefLabel
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@en
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@nl
P2860
P50
P356
P1476
The folding of the hepatitis C ...... he 3'-end of the viral genome.
@en
P2093
Alicia Barroso-Deljesus
Cristina Romero-López
P2860
P304
11697-11713
P356
10.1093/NAR/GKS927
P577
2012-10-12T00:00:00Z