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Comparative Genome Analysis of Trichophyton rubrum and Related Dermatophytes Reveals Candidate Genes Involved in InfectionFungi on the skin: dermatophytes and MalasseziaCytoplasmic localization of sterol transcription factors Upc2p and Ecm22p in S. cerevisiae.Azole drugs are imported by facilitated diffusion in Candida albicans and other pathogenic fungiZinc finger transcription factors displaced SREBP proteins as the major Sterol regulators during Saccharomycotina evolutionGenetic Basis of Antifungal Drug ResistanceClinical, cellular, and molecular factors that contribute to antifungal drug resistance.In vivo analysis of secreted aspartyl proteinase expression in human oral candidiasisThe role of Candida albicans homologous recombination factors Rad54 and Rdh54 in DNA damage sensitivity.A foot in the door for dermatophyte research.Overexpression of TUF1 restores respiratory growth and fluconazole sensitivity to a Cryptococcus neoformans vad1Delta mutant.Comparative and functional genomics provide insights into the pathogenicity of dermatophytic fungiThree small RNAs within the 10 kb trypanosome rRNA transcription unit are analogous to domain VII of other eukaryotic 28S rRNAs.Increased mRNA levels of ERG16, CDR, and MDR1 correlate with increases in azole resistance in Candida albicans isolates from a patient infected with human immunodeficiency virusThe evolution of drug resistance in clinical isolates of Candida albicansDermatophyte virulence factors: identifying and analyzing genes that may contribute to chronic or acute skin infectionsCandida albicans secreted aspartyl proteinases: isoenzyme pattern is determined by cell type, and levels are determined by environmental factors.Triclosan antagonizes fluconazole activity against Candida albicans.Azole drug import into the pathogenic fungus Aspergillus fumigatus.A flucytosine-responsive Mbp1/Swi4-like protein, Mbs1, plays pleiotropic roles in antifungal drug resistance, stress response, and virulence of Cryptococcus neoformans.RNA end-labeling and RNA ligase activities can produce a circular rRNA in whole cell extracts from trypanosomes.Comparison of sterol import under aerobic and anaerobic conditions in three fungal species, Candida albicans, Candida glabrata, and Saccharomyces cerevisiae.Methylation site within a facultatively persistent sequence in the macronucleus of Tetrahymena thermophilaCharacterization of caspofungin susceptibilities by broth and agar in Candida albicans clinical isolates with characterized mechanisms of azole resistanceA Combination Fluorescence Assay Demonstrates Increased Efflux Pump Activity as a Resistance Mechanism in Azole-Resistant Vaginal Candida albicans Isolates.Medically important fungi respond to azole drugs: an update.The R467K amino acid substitution in Candida albicans sterol 14alpha-demethylase causes drug resistance through reduced affinity.Transcriptional analyses of antifungal drug resistance in Candida albicans.Inducible azole resistance associated with a heterogeneous phenotype in Candida albicans.The "universal" leucine codon CTG in the secreted aspartyl proteinase 1 (SAP1) gene of Candida albicans encodes a serine in vivo.A fourth secreted aspartyl proteinase gene (SAP4) and a CARE2 repetitive element are located upstream of the SAP1 gene in Candida albicans.RNA dependent RNA polymerase activity associated with the double-stranded RNA virus of Giardia lamblia.The Candida albicans lanosterol 14-alpha-demethylase (ERG11) gene promoter is maximally induced after prolonged growth with antifungal drugsDermatophytes activate skin keratinocytes via mitogen-activated protein kinase signaling and induce immune responsesAn A643V amino acid substitution in Upc2p contributes to azole resistance in well-characterized clinical isolates of Candida albicans.Sequenced dermatophyte strains: growth rate, conidiation, drug susceptibilities, and virulence in an invertebrate modelCandida albicans UPC2 is transcriptionally induced in response to antifungal drugs and anaerobicity through Upc2p-dependent and -independent mechanisms.The UPC2 promoter in Candida albicans contains two cis-acting elements that bind directly to Upc2p, resulting in transcriptional autoregulation.Discovery of cryptic polyketide metabolites from dermatophytes using heterologous expression in Aspergillus nidulans.cis-Acting elements within the Candida albicans ERG11 promoter mediate the azole response through transcription factor Upc2p.
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Theodore C White
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0000-0003-3229-1378