An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress.
about
Diseases associated with defective responses to DNA damageMitochondrial DNA damage induced autophagy, cell death, and diseaseDNA repair diseases: What do they tell us about cancer and aging?UVSSA and USP7, a new couple in transcription-coupled DNA repairOxidative stress and mitochondrial dysfunction across broad-ranging pathologies: toward mitochondria-targeted clinical strategies.Mitochondrial reactive oxygen species are scavenged by Cockayne syndrome B protein in human fibroblasts without nuclear DNA damageOxidative DNA damage and nucleotide excision repair.Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic diseaseActive transcriptomic and proteomic reprogramming in the C. elegans nucleotide excision repair mutant xpa-1.Comet-FISH with strand-specific probes reveals transcription-coupled repair of 8-oxoGuanine in human cells.Reversal of mitochondrial defects with CSB-dependent serine protease inhibitors in patient cells of the progeroid Cockayne syndromeCockayne syndrome-derived neurons display reduced synapse density and altered neural network synchronyThe CSB chromatin remodeler and CTCF architectural protein cooperate in response to oxidative stress.Mitochondrial deficiency in Cockayne syndrome.Cockayne syndrome group A and B proteins converge on transcription-linked resolution of non-B DNA.Sources and consequences of oxidative damage from mitochondria and neurotransmitter signaling.Cockayne syndrome: Clinical features, model systems and pathways.Mechanistic and biological considerations of oxidatively damaged DNA for helicase-dependent pathways of nucleic acid metabolism.The fine tuning of metabolism, autophagy and differentiation during in vitro myogenesis.Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress.Overexpression of parkin rescues the defective mitochondrial phenotype and the increased apoptosis of Cockayne Syndrome A cells.Dendritic spine density is altered in a mouse model of Cockayne syndrome.2-Deoxy glucose regulate MMP-9 in a SIRT-1 dependent and NFkB independent mechanism.Nucleotide Excision Repair: From Neurodegeneration to Cancer.CSA and CSB play a role in the response to DNA breaks.miR-200a Modulates the Expression of the DNA Repair Protein OGG1 Playing a Role in Aging of Primary Human Keratinocytes.
P2860
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P2860
An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
An altered redox balance media ...... broblasts to oxidative stress.
@en
An altered redox balance media ...... broblasts to oxidative stress.
@nl
type
label
An altered redox balance media ...... broblasts to oxidative stress.
@en
An altered redox balance media ...... broblasts to oxidative stress.
@nl
prefLabel
An altered redox balance media ...... broblasts to oxidative stress.
@en
An altered redox balance media ...... broblasts to oxidative stress.
@nl
P2093
P2860
P1433
P1476
An altered redox balance media ...... ibroblasts to oxidative stress
@en
P2093
Angelo Calcagnile
Bennett Van Houten
Bratislav M Janjic
Eugenia Dogliotti
Franca Podo
Ivano Iavarone
Laura Narciso
Mariarosaria D'Errico
Miria Stefanini
P2860
P304
P356
10.1111/J.1474-9726.2012.00815.X
P577
2012-04-05T00:00:00Z