Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells - sprookjes - yvandenbroek - TriplyDB

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

http://www.wikidata.org/entity/Q42595768

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Mitral valve disease--morphology and mechanisms.Regulation of valve endothelial cell vasculogenic network architectures with ROCK and Rac inhibitors.Ligand-specific function of transforming growth factor beta in epithelial-mesenchymal transition in heart development Fine-tuning vascular fate during endothelial-mesenchymal transition.Thymosin beta4 regulates cardiac valve formation via endothelial-mesenchymal transformation in zebrafish embryos.Differential functions of genes regulated by VEGF-NFATc1 signaling pathway in the migration of pulmonary valve endothelial cells Drosophila Abelson kinase mediates cell invasion and proliferation through two distinct MAPK pathways.Loss of β-catenin promotes chondrogenic differentiation of aortic valve interstitial cells Cyclic strain induces dual-mode endothelial-mesenchymal transformation of the cardiac valve.Laminin Peptide-Immobilized Hydrogels Modulate Valve Endothelial Cell Hemostatic Regulation.Valvular interstitial cells suppress calcification of valvular endothelial cells.Inflammatory cytokines promote mesenchymal transformation in embryonic and adult valve endothelial cells The contribution of cellular mechanotransduction to cardiomyocyte form and function.Cardiac repair and regeneration: the Rubik's cube of cell therapy for heart disease.Active adaptation of the tethered mitral valve: insights into a compensatory mechanism for functional mitral regurgitation.Heterogeneous susceptibility of valve endothelial cells to mesenchymal transformation in response to TNFα.Progenitor cells confer plasticity to cardiac valve endothelium.Alternative origins of stroma in normal organs and disease.Mitral valve endothelial cells with osteogenic differentiation potential.Soluble VEGFR1 reverses BMP2 inhibition of intramembranous ossification during healing of cortical bone defects.Silencing of hHS6ST2 inhibits progression of pancreatic cancer through inhibition of Notch signalling.Long form of latent TGF-β binding protein 1 (Ltbp1L) regulates cardiac valve development.Mesenchymal state of intimal cells may explain higher propensity to ascending aortic aneurysm in bicuspid aortic valves.Developmental vascular regression is regulated by a Wnt/β-catenin, MYC, P21 (CDKN1A) pathway that controls cell proliferation and cell death.Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis.

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Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

http://www.wikidata.org/entity/Q42595768

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

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Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

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type

label

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

@en

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

@nl

prefLabel

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

@en

Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

@nl

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J.BBRC.2008.07.057

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Biochemical and Biophysical Research Communications

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Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cells

@en

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Jeong-Hee Yang

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Jill Wylie-Sears

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Jagged1-mediated Notch activation induces epithelial-to-mesenchymal transition through Slug-induced repression of E-cadherin.

Mutations in NOTCH1 cause aortic valve disease

Regulation of Notch1 and Dll4 by vascular endothelial growth factor in arterial endothelial cells: implications for modulating arteriogenesis and angiogenesis

A null mutation of Hhex results in abnormal cardiac development, defective vasculogenesis and elevated Vegfa levels

Notch promotes epithelial-mesenchymal transition during cardiac development and oncogenic transformation

A field of myocardial-endocardial NFAT signaling underlies heart valve morphogenesis

Human pulmonary valve progenitor cells exhibit endothelial/mesenchymal plasticity in response to vascular endothelial growth factor-A and transforming growth factor-beta2.

Heart valve development: endothelial cell signaling and differentiation

Aortic valve endothelial cells undergo transforming growth factor-beta-mediated and non-transforming growth factor-beta-mediated transdifferentiation in vitro

Cell biology of cardiac cushion development.

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512-516

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10.1016/J.BBRC.2008.07.057

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