Development of behavioural dysfunctions in accelerated-senescence OXYS rats is associated with early postnatal alterations in brain phosphate metabolism.
about
Senescence-accelerated OXYS rats: a model of age-related cognitive decline with relevance to abnormalities in Alzheimer disease.The mitochondria-targeted antioxidant SkQ1 but not N-acetylcysteine reverses aging-related biomarkers in rats.Rapamycin suppresses brain aging in senescence-accelerated OXYS rats.Mitochondria-targeted antioxidant SkQ1 inhibits age-dependent involution of the thymus in normal and senescence-prone rats.Alterations of retinal pigment epithelium cause AMD-like retinopathy in senescence-accelerated OXYS ratsEvaluation of effects of histochrome and mexidol on structural and functional characteristics of the brain in senescence-accelerated OXYS rats by magnetic resonance imaging.
P2860
Development of behavioural dysfunctions in accelerated-senescence OXYS rats is associated with early postnatal alterations in brain phosphate metabolism.
description
2005 nî lūn-bûn
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2005年の論文
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2005年論文
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2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
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2005年论文
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name
Development of behavioural dys ...... in brain phosphate metabolism.
@en
Development of behavioural dys ...... in brain phosphate metabolism.
@nl
type
label
Development of behavioural dys ...... in brain phosphate metabolism.
@en
Development of behavioural dys ...... in brain phosphate metabolism.
@nl
prefLabel
Development of behavioural dys ...... in brain phosphate metabolism.
@en
Development of behavioural dys ...... in brain phosphate metabolism.
@nl
P50
P1476
Development of behavioural dys ...... in brain phosphate metabolism
@en
P2093
Korbolina E
P304
P356
10.1016/J.EXGER.2005.10.009
P577
2005-11-28T00:00:00Z