about
A highly precise and portable genome engineering method allows comparison of mutational effects across bacterial speciesSystem-level genome editing in microbes.Phenotypic heterogeneity promotes adaptive evolution.Conditional DNA repair mutants enable highly precise genome engineering.Perturbation of iron homeostasis promotes the evolution of antibiotic resistance.Genome-wide analysis captures the determinants of the antibiotic cross-resistance interaction network.A standardized workflow for surveying recombinases expands bacterial genome-editing capabilities.Genome-Wide Abolishment of Mobile Genetic Elements Using Genome Shuffling and CRISPR/Cas-Assisted MAGE Allows the Efficient Stabilization of a Bacterial Chassis.Directed evolution of multiple genomic loci allows the prediction of antibiotic resistance.CRISPR-interference based modulation of mobile genetic elements in bacteriaEnzyme promiscuity shapes evolutionary innovation and optimizationNew N-phenylpyrrolamide DNA gyrase B inhibitors: Optimization of efficacy and antibacterial activityAntibiotic-resistant bacteria show widespread collateral sensitivity to antimicrobial peptidesEnzyme promiscuity shapes adaptation to novel growth substratesCRISPR-interference-based modulation of mobile genetic elements in bacteriaSecond-generation 4,5,6,7-tetrahydrobenzo[d]thiazoles as novel DNA gyrase inhibitorsHigh-Efficiency Multi-site Genomic Editing of Pseudomonas putida through Thermoinducible ssDNA RecombineeringPhylogenetic barriers to horizontal transfer of antimicrobial peptide resistance genes in the human gut microbiotaMismatch repair hierarchy of Pseudomonas putida revealed by mutagenic ssDNA recombineering of the pyrF geneDual Escherichia coli DNA Gyrase A and B Inhibitors with Antibacterial ActivityMultiple-Site Diversification of Regulatory Sequences Enables Interspecies Operability of Genetic DevicesAn optimised series of substituted N-phenylpyrrolamides as DNA gyrase B inhibitorsLimited Evolutionary Conservation of the Phenotypic Effects of Antibiotic Resistance MutationsRapid Evolution of Reduced Susceptibility against a Balanced Dual-Targeting Antibiotic through Stepping-Stone MutationsImproved bacterial recombineering by parallelized protein discovery
P50
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P50
description
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Ákos Nyerges
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Ákos Nyerges
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Ákos Nyerges
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Ákos Nyerges
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P106
P1960
Dn8nr-UAAAAJ
P2002
AkosNyerges
P21
P31
P496
0000-0002-1581-490X
P569
2000-01-01T00:00:00Z