Skeletal muscle glucose uptake during contraction is regulated by nitric oxide and ROS independently of AMPK.
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Redox Characterization of Functioning Skeletal MuscleGeneral aspects of muscle glucose uptakeSkeletal muscle nitric oxide signaling and exercise: a focus on glucose metabolismRac1--a novel regulator of contraction-stimulated glucose uptake in skeletal muscleContraction-stimulated glucose transport in muscle is controlled by AMPK and mechanical stress but not sarcoplasmatic reticulum Ca(2+) releaseStretch-stimulated glucose transport in skeletal muscle is regulated by Rac1Attempting to Compensate for Reduced Neuronal Nitric Oxide Synthase Protein with Nitrate Supplementation Cannot Overcome Metabolic Dysfunction but Rather Has Detrimental Effects in Dystrophin-Deficient mdx Muscle.Exercise and Glycemic Control: Focus on Redox Homeostasis and Redox-Sensitive Protein Signaling.Endothelial nitric oxide synthase is central to skeletal muscle metabolic regulation and enzymatic signaling during exercise in vivo.Obesity impairs skeletal muscle AMPK signaling during exercise: role of AMPKα2 in the regulation of exercise capacity in vivo.Arginine protects muscle cells from wasting in vitro in an mTORC1-dependent and NO-independent manner.AMP-activated protein kinase (AMPK) beta1beta2 muscle null mice reveal an essential role for AMPK in maintaining mitochondrial content and glucose uptake during exercise.Cilostazol Inhibits Vascular Smooth Muscle Cell Proliferation and Reactive Oxygen Species Production through Activation of AMP-activated Protein Kinase Induced by Heme Oxygenase-1.Imaging superoxide flash and metabolism-coupled mitochondrial permeability transition in living animals.Protein kinase D1 is essential for contraction-induced glucose uptake but is not involved in fatty acid uptake into cardiomyocytes.Interactions between mitochondrial reactive oxygen species and cellular glucose metabolism.Fighting obesity: When muscle meets fat.Mitochondrial antioxidative capacity regulates muscle glucose uptake in the conscious mouse: effect of exercise and diet.Contraction stimulates muscle glucose uptake independent of atypical PKC.Sarcolemmal targeting of nNOSμ improves contractile function of mdx muscle.AMPK and Exercise: Glucose Uptake and Insulin Sensitivity.ContRac1ion-mediated glucose uptake: a central role for Rac1.Electrical stimuli release ATP to increase GLUT4 translocation and glucose uptake via PI3Kγ-Akt-AS160 in skeletal muscle cells.Protein carbonylation, mitochondrial dysfunction, and insulin resistance.The role of cherries in exercise and health.Role of nitric oxide in skeletal muscle glucose uptake during exercise.Regulation of NADPH oxidases in skeletal muscle.The effects of muscle contraction and recombinant osteocalcin on insulin sensitivity ex vivo.PT-1 selectively activates AMPK-γ1 complexes in mouse skeletal muscle, but activates all three γ subunit complexes in cultured human cells by inhibiting the respiratory chain.Role of AMP-Activated Protein Kinase for Regulating Post-exercise Insulin Sensitivity.Effect of nitrate supplementation on hepatic blood flow and glucose homeostasis: a double-blind, placebo-controlled, randomized control trial.GSM 900 MHz microwave radiation affects embryo development of Japanese quails.Mitochondria dysfunction in lung cancer-induced muscle wasting in C2C12 myotubes.Downstream mechanisms of nitric oxide-mediated skeletal muscle glucose uptake during contraction.N-Acetylcysteine infusion does not affect glucose disposal during prolonged moderate-intensity exercise in humans.Local hindlimb antioxidant infusion does not affect muscle glucose uptake during in situ contractions in rat.AMPKα2 deficiency uncovers time dependency in the regulation of contraction-induced palmitate and glucose uptake in mouse muscle.N-acetylcysteine alters substrate metabolism during high-intensity cycle exercise in well-trained humans.Role of reactive oxygen species in regulation of glucose transport in skeletal muscle during exercise.Effect of N-acetylcysteine infusion on exercise-induced modulation of insulin sensitivity and signaling pathways in human skeletal muscle.
P2860
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P2860
Skeletal muscle glucose uptake during contraction is regulated by nitric oxide and ROS independently of AMPK.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@en
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@nl
type
label
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@en
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@nl
prefLabel
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@en
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@nl
P2860
P50
P1476
Skeletal muscle glucose uptake ...... and ROS independently of AMPK.
@en
P2093
Gregory R Steinberg
P2860
P304
P356
10.1152/AJPENDO.00239.2009
P577
2009-12-15T00:00:00Z