Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
about
The structural basis for cancer treatment decisions.Basal metabolic state governs AIF-dependent growth support in pancreatic cancer cells.Bit1 knockdown contributes to growth suppression as well as the decreases of migration and invasion abilities in esophageal squamous cell carcinoma via suppressing FAK-paxillin pathway.Bit1-a potential positive regulator of epithelial-mesenchymal transition in lens epithelial cells.Apoptotic Effects of Rotundic Acid on Human Esophagus and Lung Cancer Cells.Preliminary evaluation for Bit1 as a potential biomarker for squamous cell carcinoma and adenocarcinoma of esophagus.Spontaneous Single-Copy Loss of TP53 in Human Embryonic Stem Cells Markedly Increases Cell Proliferation and Survival.
P2860
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P2860
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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name
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@en
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@nl
type
label
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@en
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@nl
prefLabel
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@en
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@nl
P2093
P2860
P1433
P1476
Implications of Bit1 and AIF overexpressions in esophageal squamous cell carcinoma.
@en
P2093
Hongtao Liu
Lirong Zhang
Shanyong Yi
Shengna Han
Tianli Fan
P2860
P2888
P304
P356
10.1007/S13277-013-1073-8
P577
2013-08-17T00:00:00Z
P6179
1042719320