Diazoxide-mediated preconditioning against apoptosis involves activation of cAMP-response element-binding protein (CREB) and NFkappaB.
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Synaptic activity becomes excitotoxic in neurons exposed to elevated levels of platelet-activating factor.Dipyridamole with low-dose aspirin augments the infarct size-limiting effects of simvastatinMyocardial ischemic protection in natural mammalian hibernation.The mitochondrial K(ATP) channel--fact or fiction?The endogenous mitochondrial complex II inhibitor malonate regulates mitochondrial ATP-sensitive potassium channels: implications for ischemic preconditioning.Mitochondrial potassium ATP channels and retinal ischemic preconditioningMitochondrial therapeutics for cardioprotection.The emerging role of dipeptidyl peptidase-4 inhibitors in cardiovascular protection: current position and perspectives.Protein kinase C epsilon delays latency until anoxic depolarization through arc expression and GluR2 internalization.Small molecule inhibition of cAMP response element binding protein in human acute myeloid leukemia cells.The Slo(w) path to identifying the mitochondrial channels responsible for ischemic protection.Activation of IL-11/STAT3 pathway in preconditioned human skeletal myoblasts blocks apoptotic cascade under oxidant stress.Stimulatory heterotrimeric GTP-binding protein inhibits hydrogen peroxide-induced apoptosis by repressing BAK induction in SH-SY5Y human neuroblastoma cells.The C. elegans mitochondrial K+(ATP) channel: a potential target for preconditioning.Apple, cherry, and blackcurrant increases nuclear factor kappa B activation in liver of transgenic mice.The myocardial infarct size-limiting effect of sitagliptin is PKA-dependent, whereas the protective effect of pioglitazone is partially dependent on PKA.Bone morphogenetic protein-2 upregulates expression and function of voltage-gated K+ channels in human pulmonary artery smooth muscle cells.Hormesis and disease resistance: activation of cellular stress response pathways.Phosphodiesterase-3 inhibition augments the myocardial infarct size-limiting effects of exenatide in mice with type 2 diabetes.Phosphodiesterase III inhibition increases cAMP levels and augments the infarct size limiting effect of a DPP-4 inhibitor in mice with type-2 diabetes mellitus.
P2860
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P2860
Diazoxide-mediated preconditioning against apoptosis involves activation of cAMP-response element-binding protein (CREB) and NFkappaB.
description
2004 nî lūn-bûn
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2004年の論文
@ja
2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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name
Diazoxide-mediated preconditio ...... g protein (CREB) and NFkappaB.
@en
Diazoxide-mediated preconditio ...... sponse element-binding protein
@nl
type
label
Diazoxide-mediated preconditio ...... g protein (CREB) and NFkappaB.
@en
Diazoxide-mediated preconditio ...... sponse element-binding protein
@nl
prefLabel
Diazoxide-mediated preconditio ...... g protein (CREB) and NFkappaB.
@en
Diazoxide-mediated preconditio ...... sponse element-binding protein
@nl
P2093
P2860
P356
P1476
Diazoxide-mediated preconditio ...... g protein (CREB) and NFkappaB.
@en
P2093
Beth Vanwinkle
Randy N Rosier
Roman A Eliseev
Thomas E Gunter
P2860
P304
46748-46754
P356
10.1074/JBC.M406217200
P407
P577
2004-08-23T00:00:00Z