Genetic ablation of P65 subunit of NF-κB in mdx mice to improve muscle physiological function.

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Genetic ablation of P65 subunit of NF-κB in mdx mice to improve muscle physiological function.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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Genetic ablation of P65 subuni ...... muscle physiological function.

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Genetic ablation of P65 subuni ...... muscle physiological function.

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label

Genetic ablation of P65 subuni ...... muscle physiological function.

@en

Genetic ablation of P65 subuni ...... muscle physiological function.

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Genetic ablation of P65 subuni ...... muscle physiological function.

@en

Genetic ablation of P65 subuni ...... muscle physiological function.

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P1476

Genetic ablation of P65 subuni ...... muscle physiological function.

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P2093

Anna T Dzuricky

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Bing Wang

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Chuanqiang Pu

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Freddie Fu

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Jian Li

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Xi Yin

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Ying Tang

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P2860

NF-κB, the first quarter-century: remarkable progress and outstanding questions

Signaling to NF-kappaB

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

rAAV6-microdystrophin preserves muscle function and extends lifespan in severely dystrophic mice.

Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophy

RhoA mediates defective stem cell function and heterotopic ossification in dystrophic muscle of mice.

Rapid depletion of muscle progenitor cells in dystrophic mdx/utrophin-/- mice.

Function and genetics of dystrophin and dystrophin-related proteins in muscle.

The dystrophin glycoprotein complex: signaling strength and integrity for the sarcolemma.

Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.

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759-767

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scholarly article

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10.1002/MUS.25517

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2016-12-20T00:00:00Z

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27997693

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NF-κB