Insulin-induced laminin expression promotes a hypercontractile airway smooth muscle phenotype.
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Emerging Comorbidities in Adult Asthma: Risks, Clinical Associations, and MechanismsMetabolic Syndrome and the LungMotility, survival, and proliferation.Adiposity, fractional exhaled nitric oxide, and asthma in U.S. children.Insulin resistance, metabolic syndrome, and lung function in US adolescents with and without asthma.Caveolins and lung functionEarly-onset obesity dysregulates pulmonary adipocytokine/insulin signaling and induces asthma-like disease in micePediatric Obesity-Related Asthma: The Role of Metabolic Dysregulation.Insulin and the lung: connecting asthma and metabolic syndrome.Airway smooth muscle in airway reactivity and remodeling: what have we learned?Emerging interface between metabolic syndrome and asthma.Fibrotic response of tissue remodeling in COPD.The many faces of asthma in obesity.Protein kinase A and the exchange protein directly activated by cAMP (Epac) modulate phenotype plasticity in human airway smooth muscle.L-thyroxine promotes a proliferative airway smooth muscle phenotype in the presence of TGF-β1.The effect of obesity, weight gain, and weight loss on asthma inception and control.High-fat diet causes increased serum insulin and glucose which synergistically lead to renal tubular lipid deposition and extracellular matrix accumulation.cAMP inhibits modulation of airway smooth muscle phenotype via the exchange protein activated by cAMP (Epac) and protein kinase AThe laminin β1-competing peptide YIGSR induces a hypercontractile, hypoproliferative airway smooth muscle phenotype in an animal model of allergic asthma.Association between HOMA-IR and Lung Function in Korean Young Adults based on the Korea National Health and Nutrition Examination Survey.Functional consequences of human airway smooth muscle phenotype plasticity.{beta}-Catenin regulates airway smooth muscle contraction.Extracellular matrix in airway smooth muscle is associated with dynamics of airway function in asthma.Is impaired glucose metabolism the missing piece in the obesity-asthma puzzle?Insulin use increases risk of asthma but metformin use reduces the risk in patients with diabetes in a Taiwanese population cohort.Glucagon-Like Peptide 1 Receptor: A Novel Pharmacological Target for Treating Human Bronchial Hyperresponsiveness.Intracellular cAMP Sensor EPAC: Physiology, Pathophysiology, and Therapeutics Development.
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Insulin-induced laminin expression promotes a hypercontractile airway smooth muscle phenotype.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@en
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@nl
type
label
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@en
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@nl
prefLabel
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@en
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@nl
P2093
P2860
P356
P1476
Insulin-induced laminin expres ...... irway smooth muscle phenotype.
@en
P2093
Bart G J Dekkers
Dedmer Schaafsma
Herman Meurs
Johan Zaagsma
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P304
P356
10.1165/RCMB.2008-0251OC
P577
2009-02-12T00:00:00Z