Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
about
Protein kinase C θ deficiency increases resistance of C57BL/6J mice to Plasmodium berghei infection-induced cerebral malaria.Age-dependent sex bias in clinical malarial disease in hypoendemic regions.Augmented particle trapping and attenuated inflammation in the liver by protective vaccination against Plasmodium chabaudi malaria.The Lymphotoxin β Receptor Is Essential for Upregulation of IFN-Induced Guanylate-Binding Proteins and Survival after Toxoplasma gondii Infection.Impact of sex differences in brain response to infection with Plasmodium berghei.Lymphotoxin alpha and tumour necrosis factor are not required for control of parasite growth, but differentially regulate cytokine production during Plasmodium chabaudi chabaudi AS infection.
P2860
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@en
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@nl
type
label
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@en
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@nl
prefLabel
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@en
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@nl
P2093
P2860
P1433
P1476
Deletion of LTbetaR augments male susceptibility to Plasmodium chabaudi.
@en
P2093
P2860
P304
P356
10.1111/J.1365-3024.2005.00763.X
P577
2005-06-01T00:00:00Z