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The allergy adjuvant effect of particles - genetic factors influence antibody and cytokine responses.Carbon nanofibers have IgE adjuvant capacity but are less potent than nanotubes in promoting allergic airway responsesCan exposure to environmental chemicals increase the risk of diabetes type 1 development?Accumulation of mineral oil saturated hydrocarbons (MOSH) in female Fischer 344 rats: Comparison with human data and consequences for risk assessment.Maternal allergen immunisation to prevent sensitisation in offspring: Th2-polarising adjuvants are more efficient than a Th1-polarising adjuvant in mice.Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and InflammationInvestigations of immunogenic, allergenic and adjuvant properties of Cry1Ab protein after intragastric exposure in a food allergy model in mice.Pulmonary phthalate exposure and asthma - is PPAR a plausible mechanistic link?Toxicogenomic profiles in relation to maternal immunotoxic exposure and immune functionality in newborns.Popliteal lymph node (PLN) assay to study adjuvant effects on respiratory allergy.Micronuclei in cord blood lymphocytes and associations with biomarkers of exposure to carcinogens and hormonally active factors, gene polymorphisms, and gene expression: the NewGeneris cohortBisphenol A Is More Potent than Phthalate Metabolites in Reducing Pancreatic β-Cell Function.Mineral oil saturated hydrocarbons (MOSH) in female Fischer 344 rats; accumulation of wax components; implications for risk assessment.Exposure of Norwegian toddlers to perfluoroalkyl substances (PFAS): The association with breastfeeding and maternal PFAS concentrations.Early life exposure to bisphenol A investigated in mouse models of airway allergy, food allergy and oral tolerance.Cord blood gene expression supports that prenatal exposure to perfluoroalkyl substances causes depressed immune functionality in early childhood.Cry1Ab protein from Bacillus thuringiensis and MON810 cry1Ab-transgenic maize exerts no adjuvant effect after airway exposure.Effect of dietary pristane and other saturated mineral oils (MOSH) on autoimmune arthritis in rats.Exposure to perfluoroundecanoic acid (PFUnDA) accelerates insulitis development in a mouse model of type 1 diabetes.Exposure to bisphenol A, but not phthalates, increases spontaneous diabetes type 1 development in NOD mice.Pre-natal exposure to perfluoroalkyl substances may be associated with altered vaccine antibody levels and immune-related health outcomes in early childhood.Transmaternal bisphenol A exposure accelerates diabetes type 1 development in NOD mice.Transcriptomic profile indicative of immunotoxic exposure: in vitro studies in peripheral blood mononuclear cells.Corrigendum to "Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and Inflammation".Evaluation of the genotoxicity of 10 selected dietary/environmental compounds with the in vitro micronucleus cytokinesis-block assay in an interlaboratory comparison.Prenatal exposure to polychlorinated biphenyls and dioxins from the maternal diet may be associated with immunosuppressive effects that persist into early childhood.Prenatal exposure to bisphenol A interferes with the development of cerebellar granule neurons in mice and chicken.Particles influence allergic responses in mice--role of gender and particle size.Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice.The capacity of particles to increase allergic sensitization is predicted by particle number and surface area, not by particle mass.In vitro cytokine release from human peripheral blood mononuclear cells in the assessment of the immunotoxic potential of chemicals.Particles from wood smoke and road traffic differently affect the innate immune system of the lung.Single-walled and multi-walled carbon nanotubes promote allergic immune responses in mice.Particle size determines activation of the innate immune system in the lung.Global gene expression analysis in cord blood reveals gender-specific differences in response to carcinogenic exposure in utero.Ambient air particles from four European cities increase the primary cellular response to allergen in the draining lymph node.Offspring IgE responses are influenced by levels of maternal IgG transferred in early life.Prenatal exposure to polychlorinated biphenyls and dioxins is associated with increased risk of wheeze and infections in infants.Suppression of allergen-specific IgE in offspring after preconceptional immunisation: maternal, paternal and genetic influences.Early life interventions to prevent allergy in the offspring: the role of maternal immunization and postnatal mucosal allergen exposure.
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Noors onderzoekster
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հետազոտող
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Unni Cecilie Nygaard
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Unni Cecilie Nygaard
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Unni Cecilie Nygaard
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Unni Cecilie Nygaard
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Unni Cecilie Nygaard
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