Bardoxolone methyl (CDDO-Me or RTA402) induces cell cycle arrest, apoptosis and autophagy via PI3K/Akt/mTOR and p38 MAPK/Erk1/2 signaling pathways in K562 cells.
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Targeting Tumor Metabolism: A New Challenge to Improve Immunotherapy.Mesenchymal stem cells are attracted to latent HIV-1-infected cells and enable virus reactivation via a non-canonical PI3K-NFκB signaling pathwayAutophagy induces transforming growth factor-β-dependent epithelial-mesenchymal transition in hepatocarcinoma cells through cAMP response element binding signalling
P2860
Bardoxolone methyl (CDDO-Me or RTA402) induces cell cycle arrest, apoptosis and autophagy via PI3K/Akt/mTOR and p38 MAPK/Erk1/2 signaling pathways in K562 cells.
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Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@en
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@nl
type
label
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@en
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@nl
prefLabel
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@en
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@nl
P2093
P2860
P1476
Bardoxolone methyl (CDDO-Me or ...... naling pathways in K562 cells.
@en
P2093
Hong-Wan Dang
Shu-Feng Zhou
Xin-Yu Wang
Xue-Hong Zhang
Yin-Xue Yang
P2860
P304
P407
P577
2017-10-15T00:00:00Z